Skip to main content
International Journal of Molecular Sciences logoLink to International Journal of Molecular Sciences
. 2026 Jul 22;27(14):6493. doi: 10.3390/ijms27146493

Correction: Orlando et al. Lactobacillus rhamnosus GG and Lactobacillus paracasei IMPC2.1 Mitigate LPS-Induced Epithelial Barrier Dysfunction: A Focus on Autophagy Regulation. Int. J. Mol. Sci. 2025, 26, 11148

Antonella Orlando 1,†, Fatima Maqoud 1,†, Domenica Mallardi 1, Simona Drago 1, Eleonora Malerba 1, Guglielmina Chimienti 2, Francesco Russo 1,*
PMCID: PMC13410321  PMID: 42511883

In the original publication [1], reference [41] has been retracted and therefore requires revision.

The previous reference:

41. Wang, Y.; Kong, D. MicroRNA-136 Promotes Lipopolysaccharide-Induced ATDC5 Cell Injury and Inflammatory Cytokine Expression by Targeting Myeloid Cell Leukemia 1. J. Cell Biochem. 2018, 119, 9316–9326.

has been replaced with:

41. Ghosh, S.S.; Wang, J.; Yannie, P.J.; Ghosh, S. Intestinal Barrier Dysfunction, LPS Translocation, and Disease Development. J. Endocr. Soc. 2020, 4, bvz039.

The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Footnotes

Disclaimer/Publisher’s Note: The statements, opinions and data contained in all publications are solely those of the individual author(s) and contributor(s) and not of MDPI and/or the editor(s). MDPI and/or the editor(s) disclaim responsibility for any injury to people or property resulting from any ideas, methods, instructions or products referred to in the content.

Reference

  • 1.Orlando A., Maqoud F., Mallardi D., Drago S., Malerba E., Chimienti G., Russo F. Lactobacillus rhamnosus GG and Lactobacillus paracasei IMPC2.1 Mitigate LPS-Induced Epithelial Barrier Dysfunction: A Focus on Autophagy Regulation. Int. J. Mol. Sci. 2025;26:11148. doi: 10.3390/ijms262211148. [DOI] [PMC free article] [PubMed] [Google Scholar]

Articles from International Journal of Molecular Sciences are provided here courtesy of Multidisciplinary Digital Publishing Institute (MDPI)

RESOURCES