Volume 22, no. 13, p. 4450-4462, 2002. We inadvertently did not reference a study by Georges et al. (S. A. Georges, W. L. Kraus, K. Luger, J. K. Nyborg, and P. J. Laybourn, Mol. Cell. Biol. 22:127-137, 2002) which analyzed the effect of p300 on Tax transactivation of a reconstituted chromatin template containing the Tax-responsive 21-bp repeats. The paper was published after submission of our manuscript, and, in oversight, the reference was not added to the revised manuscript.
. 2002 Oct;22(19):6930. doi: 10.1128/MCB.22.19.6930.2002
Acetylation of Nucleosomal Histones by p300 Facilitates Transcriptionfrom Tax-Responsive Human T-Cell Leukemia Virus Type 1Chromatin Template
Hanxin Lu
1, Cynthia A Pise-Masison
1, Terace M Fletcher
1, R Louis Schiltz
1, Akhilesh K Nagaich
1, Michael Radonovich
1, Gordon Hager
1, Philip A Cole
1, John N Brady
1
Hanxin Lu
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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Cynthia A Pise-Masison
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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Terace M Fletcher
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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R Louis Schiltz
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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Akhilesh K Nagaich
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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Michael Radonovich
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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Gordon Hager
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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Philip A Cole
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
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John N Brady
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
Find articles by John N Brady
1Virus Tumor Biology Section, Basic Research Laboratory, and Laboratory of Receptor Biology and Gene Expression, National Cancer Institute, and Muscle Gene Expression Group, Laboratory of Muscle Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892; Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, Maryland 21205; and Graduate Genetics Program, Institute for Biomedical Sciences, The George Washington University, Washington, D.C. 20037
Copyright © 2002, American Society for Microbiology
PMCID: PMC135485
This corrects the article "Acetylation of Nucleosomal Histones by p300 Facilitates Transcription from Tax-Responsive Human T-Cell Leukemia Virus Type 1 Chromatin Template" on page 4450.