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. 2002 Oct;22(19):6930. doi: 10.1128/IAI.22.19.6930.2002

A Two-Stage, p16INK4A- and p53-Dependent Keratinocyte Senescence Mechanism That Limits Replicative Potential Independent of Telomere Status

James G Rheinwald 1, William C Hahn 1, Matthew R Ramsey 1, Jenny Y Wu 1, Zongyou Guo 1, Hensin Tsao 1, Michele De Luca 1, Caterina Catricalà 1, Kathleen M O'Toole 1
PMCID: PMC135486

Volume 22, no. 14, p. 5157-5172, 2002. Page 5158, column 1, Materials and Methods, “Culture media and methods,” line 5: “Flavin alenine dinucleotide (FAD) medium” should read “FAD medium.”

Page 5158, column 1, Materials and Methods, “Culture media and methods,” line 9: “10 × 10−10 M” should read “10−10 M.”

Page 5171, Acknowledgments, the first paragraph should appear as shown below.

Acknowledgments

 We thank Karl Münger for the p53DD retroviral vector, Denise Galloway for the HPV16 E6E7 retroviral vector, and Jim Koh, Chidi Ngwu, Ed Harlow, Jiri Bartek, Fiona Watt, and Cindy Loomis for providing antibodies. The development of the corneal and conjunctival cell lines was done in collaboration with Ilene Gipson. We thank James Rocco for helpful discussions about p14ARF and are grateful to Jen Benwood, Allan Tulloch, and Denise Long-Woodward for expert technical assistance.


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