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. 2026 Oct;36(10):2187. doi: 10.1101/gr.282733.126

Corrigendum: Profiling the long noncoding RNA interaction network in the regulatory elements of target genes by chromatin in situ reverse transcription sequencing

Shilin Zhang, Yichen Wang, Lin Jia, Xue Wen, Zhonghua Du, Cong Wang, Yajing Hao, Dehai Yu, Lei Zhou, Naifei Chen, Jingcheng Chen, Huiling Chen, Hui Zhang, Ilkay Celik, Günhan Gülsoy, Jianjun Luo, Baoming Qin, Xueling Cui, Zhonghui Liu, Songling Zhang, Miguel A Esteban, Ferhat Ay, Wei Xu, Runsheng Chen, Wei Li, Andrew R Hoffman, Ji-Fan Hu, Jiuwei Cui
PMCID: PMC13629685  PMID: 42823196

Genome Research 29: 1521–1532 (2019)

The authors would like to make the following corrections to the above-mentioned article:

  1. Two typos were found in the Figure 1C and Figure 3B legends, in which “mean ± SEM” should be “mean ± SD”.

  2. In the Figure 3A schematic diagram, the representative image contained duplications that may have caused confusion. We have replaced the image and updated the figure into a more precise schematic diagram by incorporating the reprogramming workflow, cellular morphology, and pluripotency marker staining.

The revised Figure 3A legend now reads:

Figure 3. Sox2-interacting lncRNAs are associated with reprogramming. (A) Mouse fibroblasts were reprogrammed into iPS cells by lentiviral transduction of OSKM (Oct4, Sox2, Klf4, c-Myc) factors. After reprogramming, iPSC colonies acquired pluripotency-associated characteristics and were stained positive for pluripotency marker (NANOG). Bottom panels: Images of fibroblasts and iPSCs were collected during reprogramming.

These corrections do not affect the interpretation and conclusions of the study.

This article has been corrected online.

doi: 10.1101/gr.282733.126


Articles from Genome Research are provided here courtesy of Cold Spring Harbor Laboratory Press

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