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. 2000 Dec;6(12):1685–1686. doi: 10.1017/s1355838200001886
The case for the involvement of the Upf3p in programmed -1 ribosomal frameshifting.
J Dinman
1, M Ruiz-Echevarria
1, W Wang
1, S Peltz
1
J Dinman
1Department of Molecular Genetics and Microbiology, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, Piscataway 08854, USA.
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M Ruiz-Echevarria
1Department of Molecular Genetics and Microbiology, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, Piscataway 08854, USA.
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W Wang
1Department of Molecular Genetics and Microbiology, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, Piscataway 08854, USA.
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S Peltz
1Department of Molecular Genetics and Microbiology, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, Piscataway 08854, USA.
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1Department of Molecular Genetics and Microbiology, Robert Wood Johnson Medical School, University of Medicine and Dentistry of New Jersey, Piscataway 08854, USA.
PMCID: PMC1370036 PMID: 11142366
See commentary "The case against the involvement of the NMD proteins in programmed frameshifting." on page 1687.
See the article "Nonsense-mediated decay mutants do not affect programmed -1 frameshifting." on page 952.