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Proceedings of the National Academy of Sciences of the United States of America logoLink to Proceedings of the National Academy of Sciences of the United States of America
. 2002 Dec 11;99(26):17219. doi: 10.1073/pnas.0136862100

Correction

PMCID: PMC139169

IMMUNOLOGY. For the article “Negative autoregulation of BCL-6 is bypassed by genetic alterations in diffuse large B cell lymphomas,” by Xing Wang, Zhiping Li, Akira Naganuma, and B. Hilda Ye, which appeared in number 23, November 12, 2002, of Proc. Natl. Acad. Sci. USA (99, 15018–15023; First Published October 29, 2002; 10.1073/pnas.232581199), the left axis label of Fig. 6 appeared incorrectly due to a printer's error. The corrected figure and its legend appear below.

Fig 6.

Fig 6.

BCL-6 preferentially binds to the wild-type exon 1 in Ly1 cells. Both Ly1 and the control Ly7 cells were analyzed for association of BCL-6 and acetylated H3 with the exon 1 sequence. Location of the PCR primers is given with respect to exon 1 and the surrounding mutations. Various amounts of recovered genomic DNA as well as a fraction of the total chromatin input were used in PCR reactions with the BCL-6-e1 and BCL-6 i2 primer pairs. IgG, normal rabbit IgG used as control antibody; N3, polyclonal anti-BCL-6 antibody; N.C., negative control PCR reaction with no template.


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