Abstract
A recent outbreak of bacillary dysentery in The Netherlands revealed that, despite the close association of HLA-B27 with post-dysenteric or reactive arthritis (ReA), not even in one family did all HLA-B27 positive patients infected by an arthritogenic bacterium, develop ReA. This dissociation shows that additional factors beside B27 may determine susceptibility to ReA.
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- Grumet F. C., Fendly B. M., Fish L., Foung S., Engleman E. G. Monoclonal antibody (B27M2) subdividing HLA-B27. Hum Immunol. 1982 Aug;5(1):61–72. doi: 10.1016/0198-8859(82)90031-3. [DOI] [PubMed] [Google Scholar]
- Keat A. Reiter's syndrome and reactive arthritis in perspective. N Engl J Med. 1983 Dec 29;309(26):1606–1615. doi: 10.1056/NEJM198312293092604. [DOI] [PubMed] [Google Scholar]
- van der Linden J. M., Keuning J. J., Wuisman J. H., Cats A., van Rood J. J. Letter: HL-A 27 and ankylosing spondylitis. Lancet. 1975 Mar 1;1(7905):520–520. doi: 10.1016/s0140-6736(75)92861-5. [DOI] [PubMed] [Google Scholar]