Abstract
An increased glucose-induced insulin response has been observed in patients susceptible to malignant hyperpyrexia. This raises the possibility that the membrane abnormality present in the calcium-storing membranes in the muscle cell in malignant hyperpyrexia may be present also in the beta cell of the pancreas.
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Selected References
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- Alford F. P., Martin F. I., Pearson M. J. The significance and interpretation of mildly abnormal oral glucose tolerance. Diabetologia. 1971 Jun;7(3):173–180. doi: 10.1007/BF01212550. [DOI] [PubMed] [Google Scholar]
- Britt B. A., Kalow W. Malignant hyperthermia: aetiology unknown. Can Anaesth Soc J. 1970 Jul;17(4):316–330. doi: 10.1007/BF03004695. [DOI] [PubMed] [Google Scholar]
- Huff T. A., Horton E. S., Lebovitz H. E. Abnormal insulin secretion in myotonic dystrophy. N Engl J Med. 1967 Oct 19;277(16):837–841. doi: 10.1056/NEJM196710192771602. [DOI] [PubMed] [Google Scholar]
- Malaisse W. J. Insulin secretion: multifactorial regulation for a single process of release. The Minkowski award lecture delivered on September 7, 1972 before the European Association for the study of Diabetes at Madrid, Spain. Diabetologia. 1973 Jun;9(3):167–173. doi: 10.1007/BF01219778. [DOI] [PubMed] [Google Scholar]
- Malaisse W., Malaisse-Lagae F. [A possible role for calcium in the stimulus-secretion coupling for glucose-induced insulin secretion]. Acta Diabetol Lat. 1970 Sep;7 (Suppl 1):264–277. [PubMed] [Google Scholar]
- Moulds R. F., Denborough M. A. Biochemical basis of malignant hyperpyrexia. Br Med J. 1974 May 4;2(5913):241–244. doi: 10.1136/bmj.2.5913.241. [DOI] [PMC free article] [PubMed] [Google Scholar]