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. 2006 Oct 18;25(20):5036. doi: 10.1038/sj.emboj.7601372

Opposing effects of the UV lesion repair protein XPA and UV bypass polymerase η on ATR checkpoint signaling

Ryan D Bomgarden, Patrick J Lupardus, Deena V Soni, Muh-Ching Yee, James M Ford, Karlene A Cimprich
PMCID: PMC1618109

Abstract

Correction to: The EMBO Journal (2006) 25, 2605–2614. doi: 10.1038/sj.emboj.7601123


The authors have discovered an error in the above article.

In the Materials and methods section, the cells used for our experiments were the SV40 transformed XPA-deficient (GM04312) and matched complemented (GM15876A) cells from Coriell Cell Repository. The XPA mutation background was incorrectly identified as XP12R0 in the original manuscript. The XP20S mutation is what was actually used.

In addition, the XPC mutant and complemented cells that we used were the SV40-transformed lines, GM15983 (XPC mutant line XP4PA) and GM16248 (XP4PA+XPC). Both were obtained from Coriell Cell Repository and are referenced below.

Emmert S, Kobayashi N, Khan SG, Kraemer KH (2000) The xeroderma pigmentosum group C gene leads to selective repair of cyclobutane pyrimidine dimers rather than 6-4 photoproducts. Proc Natl Acad Sci USA 97: 2151–2156.

The authors apologize for any inconvenience caused.


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