Abstract
A radiofibrinogen catabolism study performed on 40 patients with glomerulonephritis has shown increased fibrinogen catabolism in active immune-complex disorders.
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Selected References
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- Dixon F. J. The pathogenesis of glomerulonephritis. Am J Med. 1968 Apr;44(4):493–498. doi: 10.1016/0002-9343(68)90050-8. [DOI] [PubMed] [Google Scholar]
- Floyd M., Shenton B., Swinney J. Haemolysis in acute nephrotoxic nephritis. Br J Exp Pathol. 1973 Dec;54(6):615–627. [PMC free article] [PubMed] [Google Scholar]
- McFarlane A. S. IN VIVO BEHAVIOR OF I-FIBRINOGEN. J Clin Invest. 1963 Mar;42(3):346–361. doi: 10.1172/JCI104721. [DOI] [PMC free article] [PubMed] [Google Scholar]
- Meltzer M., Franklin E. C. Cryoglobulinemia--a study of twenty-nine patients. I. IgG and IgM cryoglobulins and factors affecting cryoprecipitability. Am J Med. 1966 Jun;40(6):828–836. doi: 10.1016/0002-9343(66)90199-9. [DOI] [PubMed] [Google Scholar]
- Tesar J. T., Schmid F. R. Conversion of soluble immune complexes into complement-fixing aggregates by IgM-rheumatoid factor. J Immunol. 1970 Nov;105(5):1206–1214. [PubMed] [Google Scholar]
- Wardle E. N., Floyd M. Fibrinogen catabolism study of three patients with bacterial endocarditis and renal disease. Br Med J. 1973 Aug 4;3(5874):255–257. doi: 10.1136/bmj.3.5874.255. [DOI] [PMC free article] [PubMed] [Google Scholar]