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. 2000 Oct;84(4):455–460. doi: 10.1136/heart.84.4.455

Thyroid disease and the heart

A Toft 1, N Boon 1
PMCID: PMC1729436  PMID: 10995425

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Figure 1:  .

Figure 1:  

Effects of hyperthyroidism on the cardiovascular system and the possible outcomes. TBV, total blood volume; LVEDV, left ventricular end diastolic volume; LVESV, left ventricular end systolic volume; SV stroke volume; SVR systemic vascular resistance; CO, cardiac output; ↑ increased; ↓ decreased. Solid arrows indicate direct effects, and dashed arrows potential outcomes. *Features for which T3 is directly responsible.

Figure 2:  .

Figure 2:  

(A) ECG in a 48 year old woman in whom there was an exacerbation of hyperthyroidism 72 hours after treatment with iodine131. (B) The pronounced ST changes slowly resolved and the tracing was normal three months after the patient became euthyroid.

Figure 3:  .

Figure 3:  

Sequential chest x rays from a patient with longstanding hypothyroidism that was complicated by congestive cardiac failure. (A) Before treatment. Cardiomegaly was caused by a combination of dilatation of all the cardiac chambers and pericardial effusion. (B) After treatment with thyroxine for nine months. (C) Seven years later, two years after the patient has stopped taking thyroxine, against medical advice, and had re-presented to the same physician with the symptoms and signs of heart failure. Reproduced from Davidson's principles and practice of medicine, 18th ed, p 570, with permission of the publisher Churchill Livingstone.

Selected References

These references are in PubMed. This may not be the complete list of references from this article.

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