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. 1964 Oct 31;2(5417):1099–1103. doi: 10.1136/bmj.2.5417.1099

Hereditary Heinz-body Anaemia, Thrombocyttopenia, and Haemoglobinopathy (Hb Köln) in a Glasgow Family

H E Hutchison, P H Pinkerton, Patricia Waters, A S Douglas, H Lehmann, D Beale
PMCID: PMC1817028  PMID: 14198723

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Selected References

These references are in PubMed. This may not be the complete list of references from this article.

  1. BAGLIONI C. An improved method for the fingerprinting of human hemoglobin. Biochim Biophys Acta. 1961 Apr 1;48:392–396. doi: 10.1016/0006-3002(61)90490-5. [DOI] [PubMed] [Google Scholar]
  2. BARKHAN P., STEVENSON M. E., PINKER G. HAEMOGLOBIN LEPORE TRAIT; AN ANALYSIS OF THE ABNORMAL HAEMOGLOBIN. Br J Haematol. 1964 Oct;10:437–441. doi: 10.1111/j.1365-2141.1964.tb00720.x. [DOI] [PubMed] [Google Scholar]
  3. BEUTLER E. The hemolytic effect of primaquine and related compounds: a review. Blood. 1959 Feb;14(2):103–139. [PubMed] [Google Scholar]
  4. CRADOCK-WATSON J. E., FENTON J. C., LEHMANN H. Tris buffer for the demonstration of haemoglobin A2 by paper electrophoresis. J Clin Pathol. 1959 Jul;12:372–373. doi: 10.1136/jcp.12.4.372. [DOI] [PMC free article] [PubMed] [Google Scholar]
  5. DACIE J. V., GRIMES A. J., MEISLER A., STEINGOLD L., HEMSTED E. H., BEAVEN G. H., WHITE J. C. HEREDITARY HEINZ-BODY ANAEMIA. A REPORT OF STUDIES ON FIVE PATIENTS WITH MILD ANAEMIA. Br J Haematol. 1964 Jul;10:388–402. doi: 10.1111/j.1365-2141.1964.tb00715.x. [DOI] [PubMed] [Google Scholar]
  6. DOXIADIS S. A., FESSAS P., VALAES T. Glucose-6-phosphate dehydrogenase deficiency. A new aetiological factor of severe neonatal jaundice. Lancet. 1961 Feb 11;1(7172):297–301. doi: 10.1016/s0140-6736(61)91476-3. [DOI] [PubMed] [Google Scholar]
  7. FRICK P. G., HITZIG W. H., BETKE K. Hemoglobin Zurich. I. A new hemoglobin anomaly associated with acute hemolytic episodes with inclusion bodies after sulfonamide therapy. Blood. 1962 Sep;20:261–271. [PubMed] [Google Scholar]
  8. GRIMES A. J., MEISLER A., DACIE J. V. CONGENITAL HEINZ-BODY ANAEMIA. FURTHER EVIDENCE ON THE CAUSE OF HEINZ-BODY PRODUCTION IN RED CELLS. Br J Haematol. 1964 Jul;10:281–290. doi: 10.1111/j.1365-2141.1964.tb00704.x. [DOI] [PubMed] [Google Scholar]
  9. HUEHNS E. R., SHOOTER E. M., BEAVEN G. H. On the recombination of canine and human haemoglobins. J Mol Biol. 1962 May;4:323–328. doi: 10.1016/s0022-2836(62)80012-6. [DOI] [PubMed] [Google Scholar]
  10. HUTCHISON H. E., JACKSON J. M., CASSIDY P. Phenacetin-induced haemolytic anaemia. Lancet. 1962 Nov 17;2(7264):1022–1024. doi: 10.1016/s0140-6736(62)92705-8. [DOI] [PubMed] [Google Scholar]
  11. HUTCHISON H. E., PINKERTON P. H., AITON M., CASSIDY P. A method for the estimation of the concentration of haemoglobin variants with particular reference to haemoglobins A2 and S and the recognition of thalassaemia minor. Scott Med J. 1963 Apr;8:149–152. doi: 10.1177/003693306300800404. [DOI] [PubMed] [Google Scholar]
  12. INGRAM V. M. Abnormal human haemoglobins. I. The comparison of normal human and sickle-cell haemoglobins by fingerprinting. Biochim Biophys Acta. 1958 Jun;28(3):539–545. doi: 10.1016/0006-3002(58)90516-x. [DOI] [PubMed] [Google Scholar]
  13. ITANO H. A., ROBINSON E. Properties and inheritance of haemoglobin by asymmetric recombination. Nature. 1959 Nov 7;184:1468–1469. doi: 10.1038/1841468a0. [DOI] [PubMed] [Google Scholar]
  14. LANGE R. D., AKEROYD J. H. Congenital hemolytic anemia with abnormal pigment metabolism and red cell inclusion bodies: a new clinical syndrome. Blood. 1958 Oct;13(10):950–958. [PubMed] [Google Scholar]
  15. MOLLISON P. L., VEALL N. The use of the isotope 51Cr as a label for red cells. Br J Haematol. 1955 Jan;1(1):62–74. doi: 10.1111/j.1365-2141.1955.tb05489.x. [DOI] [PubMed] [Google Scholar]
  16. MOZZICONACCI P., ATTAL C., PHAM-HUU-TRUNG, MALASSENET R., BESSIS M. [A new case of congenital hemolytic anemia with intra-erythrocytic inclusions and dark urines. The importance of splenectomy in the appearance of these inclusions]. Nouv Rev Fr Hematol. 1961 Nov-Dec;1:832–846. [PubMed] [Google Scholar]
  17. POULIK M. D. Starch gel electrophoresis in a discontinous system of buffers. Nature. 1957 Dec 28;180(4600):1477–1479. doi: 10.1038/1801477a0. [DOI] [PubMed] [Google Scholar]
  18. ROBINSON A. R., ROBSON M., HARRISON A. P., ZUELZER W. W. A new technique for differentiation of hemoglobin. J Lab Clin Med. 1957 Nov;50(5):745–752. [PubMed] [Google Scholar]
  19. SCOTT J. L., HAUT A., CARTWRIGHT G. E., WINTROBE M. M. Congenital hemolytic disease associated with red cell inclusion bodies, abnormal pigment metabolism and an electrophoretic hemoglobin abnormality. Blood. 1960 Sep;16:1239–1252. [PubMed] [Google Scholar]
  20. SELWYN J. G., DACIE J. V. Autohemolysis and other changes resulting from the incubation in vitro of red cells from patients with congenital hemolytic anemia. Blood. 1954 May;9(5):414–438. [PubMed] [Google Scholar]
  21. SHIBATA S., IUCHI I., MIYAJI T., UEDA S., TAKEDA I. HEMOLYTIC DISEASE ASSOCIATED WITH THE PRODUCTION OF ABNORMAL HEMOGLOBIN AND INTRAERYTHROCYTIC HEINZ BODIES. Nihon Ketsueki Gakkai Zasshi. 1963 Apr;26:164–173. [PubMed] [Google Scholar]
  22. SINGER K., CHERNOFF A. I., SINGER L. Studies on abnormal hemoglobins. I. Their demonstration in sickle cell anemia and other hematologic disorders by means of alkali denaturation. Blood. 1951 May;6(5):413–428. [PubMed] [Google Scholar]
  23. WENT L. N., MACIVER J. E. Thalassemia in the West Indies. Blood. 1961 Feb;17:166–181. [PubMed] [Google Scholar]
  24. WORMS R., BERNARD J., BESSIS M., MALASSENET R. [Congenital hemolytic anemia with intra-erythrocytic inclusions and dark urines. Report of a new case with electron microscope study]. Nouv Rev Fr Hematol. 1961 Nov-Dec;1:805–818. [PubMed] [Google Scholar]

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