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. 1976 Nov 20;115(10):1016–1019.

Possible mechanisms of normal amylase activity in hyperlipemic pancreatitis.

S Mishkin, J Bates, J O'Hashi, P Schneider, A D Sniderman, R O Wolf
PMCID: PMC1878885  PMID: 206333

Abstract

Lipemic serum from three patients with acute pancreatitis and type IV hyperlipemia was fractionated into very-low-density lipoproteins and clear serum. Amylase activity (determined by the Phadebas method) in the component fractions did not exceed that in the original lipemic serum. Addition of these fractions or VLDL and chylomicrons from asymptomatic patients with hyperlipemia to nonlipemic serum from patients with "routine acute pancreatitis" did not inhibit amylase activity or alter the electrophoretic mobility of amylase isoenzymes. Therefore the normal amylase activity often observed in hyperlipemic pancreatitis does not result from an inhibition of amylase activity by serum lipoproteins.

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Selected References

These references are in PubMed. This may not be the complete list of references from this article.

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