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The Journal of Experimental Medicine logoLink to The Journal of Experimental Medicine
. 1954 Jan 31;99(2):119–124. doi: 10.1084/jem.99.2.119

THE MECHANISM OF ALLOXAN PROTECTION IN EXPERIMENTAL ATHEROSCLEROSIS

D L Cook 1, Lois M Mills 1, D M Green 1
PMCID: PMC2180343  PMID: 13130788

Abstract

Experiments were performed to compare the effects of cholesterol feeding in (a) control rabbits, (b) alloxan-diabetic rabbits, and (c) rabbits injected with alloxan while the pancreas was temporarily occluded from the circulation. The alloxan-diabetic rabbits consumed significantly higher quantities of cholesterol and food and had serum cholesterol and lipoprotein (Sf 5–9 and Sf 16–30) concentrations significantly increased over the control levels. They failed to show a commensurate increase in the degree of atherosclerosis. Rabbits in which the diabetogenic action of alloxan was prevented by temporary occlusion of the pancreas from the circulation during its administration developed grades of hypercholesterolemia, hyperlipoproteinemia, and atherosclerosis not significantly different from the controls. The results are interpreted as indicating that the effects of alloxan on tissues other than the pancreas do not protect against experimental atherosclerosis produced by cholesterol feeding.

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Selected References

These references are in PubMed. This may not be the complete list of references from this article.

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