Skip to main content
The Journal of Experimental Medicine logoLink to The Journal of Experimental Medicine
. 1986 Sep 1;164(3):932–937. doi: 10.1084/jem.164.3.932

Cytotoxic T lymphocytes against disease-associated determinant(s) in ankylosing spondylitis

PMCID: PMC2188396  PMID: 3528379

Abstract

Cytotoxic T lymphocytes, induced by stimulating the PBMC of an HLA-B27+ normal individual (B27+, AS-) with the PBMC of an HLA-identical sibling suffering from ankylosing spondylitis (AS) (B27+, AS+), specifically lyse B27+, AS+ PBMC but not PBMC from HLA-27+ or B27-, AS- normal controls, or from HLA-B27- AS patients (B27-,AS+). CTL of similar specificity can also be raised by immunizing in vitro B27+,AS- cells with autologous cells modified by cross-reactive bacterial antigens. These results suggest that CTL can recognize certain bacterial antigens in association with HLA-B27 and that this interaction may lead to an inflammatory episode during the initial stages of the disease.

Full Text

The Full Text of this article is available as a PDF (366.0 KB).

Selected References

These references are in PubMed. This may not be the complete list of references from this article.

  1. Archer J. R., Stubbs M. M., Currey H. L., Geczy A. F. Antiserum to Klebsiella K43 BTS 1 specifically lyses lymphocytes of HLA-B27-positive patients with ankylosing spondylitis from a London population. Lancet. 1985 Feb 9;1(8424):344–345. doi: 10.1016/s0140-6736(85)91122-5. [DOI] [PubMed] [Google Scholar]
  2. Avakian H., Welsh J., Ebringer A., Entwistle C. C. Ankylosing spondylitis, HLA-B27 and Klebsiella. II. Cross-reactivity studies with human tissue typing sera. Br J Exp Pathol. 1980 Feb;61(1):92–96. [PMC free article] [PubMed] [Google Scholar]
  3. Geczy A. F., Alexander K., Bashir H. V., Edmonds J. A factor(s) in Klebsiella culture filtrates specifically modifies an HLA-B27 associated cell-surface component. Nature. 1980 Feb 21;283(5749):782–784. doi: 10.1038/283782a0. [DOI] [PubMed] [Google Scholar]
  4. McGuigan L. E., Geczy A. F., Prendergast J. K., Edmonds J. P., Hart H. H., Bashir H. V. HLA-B27 associated cross-reactive marker on the cells of New Zealand patients with ankylosing spondylitis. Ann Rheum Dis. 1986 Feb;45(2):144–148. doi: 10.1136/ard.45.2.144. [DOI] [PMC free article] [PubMed] [Google Scholar]
  5. Prendergast J. K., McGuigan L. E., Geczy A. F., Kwong T. S., Edmonds J. P. Persistence of HLA-B27 cross-reactive bacteria in bowel flora of patients with ankylosing spondylitis. Infect Immun. 1984 Dec;46(3):686–689. doi: 10.1128/iai.46.3.686-689.1984. [DOI] [PMC free article] [PubMed] [Google Scholar]
  6. Prendergast J. K., Sullivan J. S., Geczy A., Upfold L. I., Edmonds J. P., Bashir H. V., Reiss-Levy E. Possible role of enteric organisms in the pathogenesis of ankylosing spondylitis and other seronegative arthropathies. Infect Immun. 1983 Sep;41(3):935–941. doi: 10.1128/iai.41.3.935-941.1983. [DOI] [PMC free article] [PubMed] [Google Scholar]
  7. Sullivan J., Upfold L., Geczy A. F., Bashir H. V., Edmonds J. P. Immunochemical characterization of Klebsiella antigens which specifically modify an HLA-B27-associated cell-surface component. Hum Immunol. 1982 Dec;5(4):295–307. doi: 10.1016/0198-8859(82)90021-0. [DOI] [PubMed] [Google Scholar]
  8. Vartak H. G., Rele M. V., Rao M., Deshpande V. V. A method for concentrating dilute solutions of macromolecules. Anal Biochem. 1983 Aug;133(1):260–263. doi: 10.1016/0003-2697(83)90252-x. [DOI] [PubMed] [Google Scholar]
  9. van Bohemen C. G., Grumet F. C., Zanen H. C. Identification of HLA-B27M1 and -M2 cross-reactive antigens in Klebsiella, Shigella and Yersinia. Immunology. 1984 Aug;52(4):607–610. [PMC free article] [PubMed] [Google Scholar]
  10. van Rood J. J., van Leeuwen A., Ivanyi P., Cats A., Breur-Vriesendorp B. S., Dekker-Saeys A. J., Kijlstra A., van Kregten E. Blind confirmation of Geczy factor in ankylosing spondylitis. Lancet. 1985 Oct 26;2(8461):943–944. doi: 10.1016/s0140-6736(85)90871-2. [DOI] [PubMed] [Google Scholar]

Articles from The Journal of Experimental Medicine are provided here courtesy of The Rockefeller University Press

RESOURCES