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The Journal of Experimental Medicine logoLink to The Journal of Experimental Medicine
. 2008 Mar 17;205(3):737. doi: 10.1084/jem.20062447020108c

Jinx, an MCMV susceptibility phenotype caused by disruption of Unc13d: a mouse model of type 3 familial hemophagocytic lymphohistiocytosis

Karine Crozat, Kasper Hoebe, Sophie Ugolini, Nancy A Hong, Edith Janssen, Sophie Rutschmann, Suzanne Mudd, Sosathya Sovath, Eric Vivier, Bruce Beutler
PMCID: PMC2275393

Vol. 204, No. 4, April 16, 2007. Pages 853–863.

Please note that an error appeared in Fig. 4 B, which displays the genomic sequence with the Unc13djinx point mutation. The sequence displayed in Fig. 4 B is from the minus strand (The NCBI GenBank reference assembly for the linear genomic DNA sequence of Unc13d [Locus NC_000077] is considered the plus strand). The indicated mutation is correct, but the indicated position of the resulting new donor splice site is incorrect. The correct position of the mutation and new donor splice site within intron 26, shown in the plus strand, is below:

11311 ctccaaggct cacgagtCag tggcatgttg 11340 (C57BL/6J)

11311 ctccaaggct cacgagt Aag tggcatgttg 11340 (jinx)

The sequence shown is from GenBank genomic region NC_000077 for the linear genomic DNA sequence of Unc13d. The mutated C is shown in a bold capital letter, and the new donor splice site is underlined.


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