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. 1976 May;126(2):852–860. doi: 10.1128/jb.126.2.852-860.1976

Lack of glucose phosphotransferase function in phosphofructokinase mutants of Escherichia coli.

R A Roehl, R T Vinopal
PMCID: PMC233222  PMID: 177406

Abstract

Phosphofructokinase (pfkA) mutants of Escherichia coli are impaired in growth on all carbon sources entering glycolysis at or above the level of fructose 6-phosphate (nonpermissive carbon sources), but growth is particularly slow on sugars, such as glucose, which are normally transported and phosphorylated by the phosphoenolpyruvate, (PEP)-dependent phosphotransferase system (PTS).

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Selected References

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