Skip to main content
Proceedings of the National Academy of Sciences of the United States of America logoLink to Proceedings of the National Academy of Sciences of the United States of America
. 2008 Aug 27;105(36):13696. doi: 10.1073/pnas.0807268105

Correction for Davies et al., TASK channel deletion in mice causes primary hyperaldosteronism

PMCID: PMC2533253

PHYSIOLOGY. For the article “TASK channel deletion in mice causes primary hyperaldosteronism,” by Lucinda A. Davies, Changlong Hu, Nick A. Guagliardo, Neil Sen, Xiangdong Chen, Edmund M. Talley, Robert M. Carey, Douglas A. Bayliss, and Paula Q. Barrett, which appeared in issue 6, February 12, 2008, of Proc Natl Acad Sci USA (105:2203–2208; first published February 4, 2008; 10.1073/pnas.0712000105), the authors note that on page 2204, right column, line 18, the statement “a recent study reports sex differences in effects of TASK-1 subunit deletion on adrenal development and zonation; males appear normal, and females show marked developmental abnormalities (25)” is misreferenced and should instead cite ref. 48. The added reference appears below.

References

  • 48.Heitzmann D, et al. Invalidation of TASK1 potassium channels disrupts adrenal gland zonation and mineralocorticoid homeostasis. Embo J. 2008;27:179–187. doi: 10.1038/sj.emboj.7601934. [DOI] [PMC free article] [PubMed] [Google Scholar]

Articles from Proceedings of the National Academy of Sciences of the United States of America are provided here courtesy of National Academy of Sciences

RESOURCES