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. 2009 Nov;128(3):393–404. doi: 10.1111/j.1365-2567.2009.03121.x

Table 1.

Clinical information for patients with human immunodeficiency virus 1 (HIV-1) infection

CD4+ T cells
CD4+ T-cell nadir
CD8+ T cells
Patients Sex (% male) Age (years) Viral load (log copies/ml) Count (cells/μl) Percentage Count (cells/μl) Percentage Count (cells/μl) Percentage
HAART-treated [mean ± SD (range)] 100 41 ± 13 (27–74) ≤ DL 841 ± 77 (497–1274) 34·7 ± 6·2 (22·5–45·1) 431 ± 119 (241–645) 23·6 ± 6·4 (17·9–38·0) 962 ± 418 (400–1710) 39·1 ± 11·0 (23·2–62·5)
Untreated [mean ± SD (range)] 78 37 ± 9 (26–62) 4·29 ± 0·71 (2·97–5·30) 626 ± 172 (297–996) 27·4 ± 6·8 (12·5–38·2) 471 ± 143 (210–920) 25·5 ± 8·3 (12·5–40·1) 1,189 ± 519 (381–2338) 48·8 ± 10·1 (31·1–74·0)

Thirty-six per cent of patients were successfully treated with highly active antiretroviral therapy (HAART). Of these, six of 13 patients (46%) were on a protease-inhibitor-containing HAART regimen. Two of 36 patients were coinfected with hepatitis C virus and one with hepatitis B virus. Just one patient was coinfected with both hepatitis B and C viruses. Sexual contact was the most frequently reported risk factor for acquiring HIV-1 infection (31 of 36 patients).

DL, detection limit; SD, standard deviation.