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. Author manuscript; available in PMC: 2010 Feb 14.
Published in final edited form as: J Psychiatr Ment Health Nurs. 2008 Dec;15(10):833–849. doi: 10.1111/j.1365-2850.2008.01321.x

The Relationship between Community Violence Exposure and Mental Health Symptoms in Urban Adolescents

Catherine C McDonald 1, Therese R Richmond 2
PMCID: PMC2821658  NIHMSID: NIHMS164831  PMID: 19012675

Abstract

Urban adolescents are exposed to a substantial amount of community violence which has the potential to influence psychological functioning. To examine the relationship between community violence exposure and mental health symptoms in urban adolescents, a literature review using MEDLINE, CINAHL, PubMed, PsycINFO, CSA Social Services, and CSA Sociological Abstracts was conducted. Search terms included adolescent/adolescence, violence, urban, mental health, well-being, emotional distress, depression, anxiety, posttraumatic stress disorder, and aggression. Twenty six empirical research articles from 1997–2007 met inclusion criteria for review. Findings indicate an influence of community violence exposure on mental health symptoms, particularly posttraumatic stress and aggression. Mediators and moderators for community violence exposure and mental health symptoms help explain relationships. Limitations in the literature are the lack of consistency in measurement and analysis of community violence exposure, including assessment of proximity and time frame of exposure, and in analysis of victimization and witnessing of community violence. Knowledge about identification of urban adolescents exposed to chronic community violence and who experience mental health symptoms is critical to mental health nursing practice and research.

Keywords: Adolescent, Community Violence, Mental Health, Urban


Urban adolescents report very high rates of community violence exposure (CVE); more than 85% witness some form of violence in their lifetime (Farrell & Bruce, 1997; Overstreet & Braun, 2000; Mazza & Reynolds, 1999; Pastore, Fisher, & Friedman, 1996) and as many as 69% report direct victimization (Duckworth, Hale, Clair, & Adams, 2000; Howard, Feigelman, Li, Cross, & Rachuba, 2002; Overstreet & Braun, 2000). These rates make it important to examine the effects of CVE on the psychological well-being of urban adolescents. For this paper, CVE is considered to be “deliberate acts intended to cause harm against a person or persons in the community” (Cooley, Turner, & Beidel, 1995, p. 202). CVE encompasses direct victimization and witnessing violence against others.

Depression, anxiety, posttraumatic stress disorder (PTSD), and aggression have negative associations with CVE in urban adolescents (Cooley-Quille, Boyd, Frantz, & Walsh, 2001; Foster, Kuperminc & Price, 2004; Gorman-Smith & Tolan, 1998; Mazza & Reynolds, 1999). These relationships between CVE and mental health, however, are not always consistent, and the impact of CVE on urban adolescents’ psychological functioning is not clearly established (Farrell & Bruce, 1997; White, Bruce, Farrell, & Kliewer, 1998). Some investigators discuss desensitization, where adolescents adapt by having lower than expected mental health symptoms in response to CVE (McCart et al., 2007; Ng-Mak, Salzinger, Feldman, & Stueve, 2004). These discrepancies call for a better understanding of the correlates between CVE and mental health.

The purpose of this article is to examine the research on the relationships between CVE and mental health symptoms in urban adolescents, identify major findings, and discuss gaps for future exploration. This integrative analysis aims to garner a better understanding of the effects of CVE on mental health and help to provide direction for nursing research and practice. This information can provide mental health nurses crucial information to aid in caring for the mental health needs of vulnerable and often disadvantaged adolescents.

Methods

A literature search in MEDLINE, CINAHL, PubMed, PsycINFO, CSA Social Services Abstracts, and CSA Sociological Abstracts was conducted. In the first search, keywords included: adolescent/adolescence, violence, urban, mental health, well-being, and emotional distress. The additional key terms of depression, anxiety, PTSD, and aggression were then used to target these four mental health symptoms as primary variables of interest as identified from the initial search. In order to analyze the most current literature, included in analysis were peer-reviewed, English-language journal articles on human research from 1997–2007. Empirical studies on the relationship between CVE and mental health variables were included. To be included, studies had to capture at least one of the core characteristics of CVE: direct victimization or witnessing violence in the community. Samples of adolescents in these studies had to be within the age range of 10–19 years, recruited via school or community settings, and living in or attending school in urban areas in the United States (Center for Disease Control, 2007; World Health Organization, 2004). Selecting community based samples of adolescents draws from the implication that findings from clinically referred samples may not be as generalizable as non-referred samples (Ozer, 2005; Ozer & McDonald, 2006; Ozer & Weinstein, 2004).

Excluded from analysis were studies including both urban and suburban samples; studies with urban and suburban participants do not always distinguish between settings when analyzing the relationship between CVE and mental health (e.g. Flannery, Singer, & Wester, 2003; O’Keefe, 1997). Studies with samples of refugee and homeless adolescents were excluded. Refugees may experience multiple traumas and losses, often related to war, and present with a unique set of mental health problems (Ehnholt & Yule, 2006). Homeless adolescents are particularly vulnerable to victimization in their settings and are at great risk for mental health problems (Stewart et al., 2004). Studies examining substance abuse did not fit inclusion criteria or were excluded because it was often treated as a risk behavior, not in the same context as other mental health symptoms (e.g. Albus, Weist & Perez-Smith, 2004). Twenty six articles met criteria for analysis and were examined through study sample and design, measurement of key variables, and key findings on the relationship between CVE and mental health symptoms (see Table 1).

Table 1.

Studies Examining CVE and Mental Health in Urban Adolescents

Authors Setting, Sample & Design Instruments Used to Measure CVE
and Mental Health Variables
Key Findings
(Victimization=V; Witnessing=W)
Cooley-Quille et al., 2001 Inner-city Parochial High
School (HS)
Phase I:
N=185
Ages 13–18
43.8% Female
56.2.5% Male
90% African American
Phase II:
N=33
No SES data
Cross-sectional with 2 phases
Phase I:
Violence Exp: CREV
Anxiety: STAIC Behavior
(Internalizing & Externalizing (I&E):
YSR (I & E separately)
Phase II:
Depressive Symptoms: CDI
Diagnostic Interview: ADIS-C
Fears: FSSC-R
Phase I: (Age and gender covaried)
High CVE group had more fears,
internalizing behaviors, and trait anxiety
than low CVE group. No difference in
externalizing behaviors between low
and high CVE groups.
Females with high CVE reported more
internalizing problems (withdrawal,
anxiety, depressive symptoms)-Level of
CVE did not affect males’ behaviors.
Phase II: PTSD symptoms correlated
with CVE (.42)* and V (.41)*.
Separation anxiety correlated with CVE
(.50)* and W (.56)**.
(Regression) CVE predicted PTSD
symptoms (R2=.16)*. W predicted
separation anxiety symptoms (R2=.20)*.
Dempsey, 2002 Inner-city
Elementary (ES) & Middle
(MS) Schools.
N=120
Ages 10–14
55% Female
45% Male
100% African American
Avg $6,000/yr
Cross-sectional
Violence Exp: “Things”
Distress/PTSD Symptoms: CCDS
Depressive Symptoms: CDI
Trait Anxiety: STAIC
CVE correlated with depression
(.36)***, PTSD symptoms (.45)***, and
trait anxiety (.28)**.
(Regression) CVE predictive of PTSD
(Δ.R2=.19)**, depression
(Δ.R2=.10)*** and anxiety
(Δ.R2=.08)**. Negative coping was a
mediator to PTSD (reduced predictive
ability of CVE by 38%), depression (by
47%) and anxiety (by 31%). Together,
negative coping and CVE explained
moderate degree of variance.
Duckworth et al., 2000 Urban area MS
N=181
Ages 11–15
68% Female
32% Male
100% African American
88% eligible for free lunch
program (FLP):(Less than
$9999 per year in this study)
Cross-sectional
Violence Exp: SCECV
PTS: PTSDRI-C
Behavior I&E: YSR (total score)
PTS correlated with W (.25)**,
V (.19)**, and Community Chaos (CC)
(.30)**. Problem behaviors correlated
with W (.27)**, V (.28)**, and CC
(.29)**.
(Regression) V predictive of behavior
problems (Δ.R2=.03)*. CC predictive of
PTS (Δ.R2=.02)*. CC mediated
relationship between W and PTS. CC
mediated relationship between W and
behavior problems. Full model of age,
CC, V and W accounted to 10%
variance in PTS and 12% variance in
problem behaviors.
Farrell & Bruce, 1997 Urban public MS
N=436 at 3 data points.
Ages 11–15
58% Female
42% Male
94% African American
52% eligible FLP
Longitudinal- 9/94-6/95
Violence Exp: “Things”
Emotional Distress: 3 items each from
the Depression and Anxiety subscales of
Emotional Distress scale of the WAI
Violent Behavior: BFS
CVE (W) in females and males
correlated with violent behavior (.38)**
(.40)** but not emotional distress at
Time 1.
(SEM) CVE (W) not related to changes
in depression or anxiety over time. CVE
(W) related to changes in frequency of
violent behavior in females, but not in
males.
Foster et al., 2004 Boys & Girls Clubs in a major
metropolitan area
N=146
Ages 11–16
42.5% Female
57.5% Male
“Predominately African
American” community
“Lower income”
Cross-sectional
Violence Exp: CECV Anxiety,
Depression, Anger, PTS
symptoms, & Dissociation: TSCC
V correlated with anxiety (.31)**,
depression (.33)**, anger (.59)**,
dissociation (.52)** and PTS (.30)**for
males. W correlated with anger (.30)**
and dissociation (.23)** for males. V
and W correlated with depression (.32)*
(.31)*, anger (.41)** (.39)** and
dissociation (.31)* (.32)* for females.
(Regression) W predictor in anger
(Δ.R2=.14)** and dissociation
(Δ.R2=.09)*. V predictor in anxiety
(Δ.R2=.08)**, depression (Δ.R2=.11)**
anger (Δ.R2=.27)***, PTS (Δ.R2=.07)*,
and dissociation (Δ.R2=.20)*. Gender
moderated relationship between W and
depressive symptoms and W and
anxiety. Gender did not moderate for V
and symptoms. Males more distressed
by V than W; Females had no difference
in impact of V or W.
Gorman-Smith & Tolan, 1998 5th & 6th grades in public
schools from the CYDS
N=245 at 2 data points
Ages 11–15
100% Male
African American and Latino
(% not reported)
48%<$10,000/yr
Longitudinal-1 yr
Violence Exp: Exposure to Violence
Interview (Tolan & Gorman-Smith, 1991)
Depression, anxiety, and aggression:
subscales of the CBCL completed by
adolescent, parent, and teacher
(combined standardized scores from
each source)
CVE correlated with aggression Time 1
(.22)** and Time 2 (.31)**. CVE
correlated with anxiety/depression Time
1 (.19)** and Time 2 (.21)**
(Significant interactions): CVE related
to low cohesion in family and
anxiety/depression (.30)***. CVE also
related to aggression in families with
high levels of structure (.38)***.
(Multiple regression) CVE over past
year related to current level of
depression/anxiety (Δ.R2=.03)*** and
aggression (Δ.R2=.05)***, controlling
for previous level of symptoms
Hammack et al., 2004 6th & 7th grades in six inner-
city public schools
N=196 at Time 1 and N=159 at
Time 2
6th and then 7th graders
Gender not reported
100% African American
Median between $10,000-
$20,000
Longitudinal-1 yr
Violence Exp: “My Exposure to
Violence Interview”
Anxiety: STAIC
Depressive Symptoms: CDI
W correlated with depressive symptoms
at Time 1 (.13)* & Time 2 (.16)* for
females only. V correlated with
depressive symptoms at Time 1 (.14)*
& Time 2 (.18)*.
(Regression) Anxiety at Time 1:
Protective-stabilizing effects for W and
maternal closeness (Δ.R2=.02)* and W
and social support in females
(Δ.R2=.02)*. At Time 2: Promotive-
reactive for W and time with family for
females (Δ.R2=.02)* and for W and V
and social support in females (both
Δ.R2=.02)*.
Depressive symptoms at Time 1:
Protective-stabilizing for V and social
support (Δ.R2=.02)*. Promotive-
reactive effects for W and time with
family (Δ.R2=.04)** and V and daily
support for females (Δ.R2=.05)**.
Protective-stabilizing effects for V and
daily support for males (Δ.R2=.05)**
and for W and daily support for males
(Δ.R2=.04)**. At Time 2: Promotive-
reactive for V and maternal closeness
for males (Δ.R2=.03)*.
Howard et al., 2002 Community based-Low-
income public housing in city
N=349
Ages 9–15
44% Female
56% Male
96% African American
Low- income
Cross-sectional
Violence Exp: SCECV
Distress: CCDS
V and W each related to intrusive
thoughts (.16)**(.26)***, distraction
(.12)*(.24)** and lack of belongingness
(.26)*** (.19)**.
(Regression) W part of predictive model
for intrusive thoughts,
vigilance/avoidance, and distraction. V
part of predictive model for
despondency about the future and lack
of belongingness.
Mazza & Reynolds, 1999 Inner-city Parochial 6th–8th
N=94
Ages 11–15
61% Female
39% Male
70% African American
22% Hispanic
School in ‘Low-income area’
Cross-sectional
Violence Exp: EVQ
Depression: RADS
Suicidal Ideation: SIQ-J
PTS: APS-PTS
For females and males, measures of
CVE correlated with RADS (.29)*
(.51)**, SIQ-J (.39)**(.42)**, and APS-
PTSDS (.51)***(.62)***.
(Regression) CVE did not add to
depression severity or suicidal ideation
when other variables controlled. CVE
predicted PTSD symptomatology
(Δ.R2=.07)***.
PTSD symptomatology functioned as a
mediator between CVE and depression
and suicidal ideation.
McCart et al., 2007 Telephone survey of urban-
dwelling youth
N=1245
Ages 12–17
51% Female
49% Male
53% White (Non-Hispanic),
27% African American, 15%
Hispanic, 3% Native
American, 2% Asian American
No data on SES
Cross-sectional
Violence Exp: Structured Interview
from the National Survey of
Adolescents
PTSD: NWS-PTSD Module
Delinquency: Modified SRDS
CVE correlated with PTSD symptoms
(.32)*** and delinquency (.38)***.
(Regression-Model also included
indicators for family violence and
sexual assault in the same step as CVE)
CVE had significant linear effects on
PTSD symptoms for males
(Δ.R2=.12)*** and females
(Δ.R2=.12)***. No curvilinear effects
of CVE and PTSD symptoms in males,
but small negative curvilinear effects for
females (Δ.R2=.02)**.
CVE had a linear effect on delinquency
for males (Δ.R2=.14)***. CVE had a
significant positive curvilinear effect on
delinquency for females (Δ.R2=.03)**.
McGee et al., 2001 Census tract data from school,
church & community org
servicing inner-city youth
N=306
Ages 12–18
49% Female
51% Male
86% African American
No data on SES
Cross-sectional
Violence Exp: SCECV
Depression: CDI
Anxiety: RCMAS
Delinquency: Indicators developed by
Jang & Thornberry (1998)
Females exposed to CVE were more
likely to exhibit anxiety or depressive
symptoms than males exposed to CVE.
Moses, 1999 City Public HS
N=337
Ages 14–19
62% Female
38% Male
44.5% African American
50.9% Hispanic
‘From low SES
neighborhoods’
Cross-sectional
Violence Exp: 6 questions from a non-
published instrument.
Depression and Hostility: Scales of the
SCL-90-R
W violence against family (.11)* and
rape (.21)*** correlated with depression
scores. Being shot/stabbed (.23)***,
beaten up/jumped (.14)**, rape
(.17)***, W violence against friends
(.20)***, and W violence against family
(.21)*** all correlated with hostility.
Total CVE correlated with depression
for females (.14)*, but not for males. W
violence against family correlated with
depression scores for males (.20)*.
(Regression) CVE predicted hostility in
males (adj.R2 .11)*** and
females (adj.R2 .07)***. CVE predicted
depression in females (adj.R2 .08)*
Ng-Mak et al., 2004 Inner-city 6th grade
N=471
Mean age 12.3,
51% Female
49% Male
34.6% African American
62.2% Hispanic
59% on public assistance
Cross-sectional
Violence Exp: SCEVC
Distress: 3 items from CICS and Parent
report of CBCL (parent report and
adolescent report scored separately)
Aggression: PCAP and Parent report of
items from the CBCL
(Regression) Linear association between
CVE and distress self-report symptoms
(Δ.R2=.07)***.
Linear association between CVE and
aggressive behavior self report
(Δ.R2=.16)*** and parent report
(Δ.R2=.03)***. Quadratic effect of
CVE on distress self-report symptoms
(Δ.R2=.01)**.
Overstreet & Braun, 2000 Inner-city free Summer camp
for 5th–8th graders
N=70
Ages 10–15
54% Female
46% Male
100% African American
Avg $6,000/yr
Cross-sectional
Violence Exp: Exposure to Community
Violence (ECV), a modified version of
“Things”
Distress/PTSD: CCDS
CVE related to PTS symptomatology
(.40)
***. (Regression) Decreased neighborhood
safety and increased family conflict
mediated impact of CVE on PTS
symptoms.
Overstreet et al., 1999 Inner-city free Summer camp
for 5th–8th graders
N=75
Ages 10–15
52% Female
48% Male
100% African American
Avg $6,000/yr
Cross-sectional
Violence Exp.: ECV A modified version
of “Things”
PTSD: CCDS
Depression: CDI
CVE correlated with PTSD symptoms
(.41)***
(Regression) CVE contributed to
prediction of PTSD (CVE +Family
Support Δ.R2=.28) and depressive
symptoms (CVE + Family Support
Δ.R2=.07). Availability of family
support did not moderate effects of CVE
on PTSD symptoms. Absence of
mother moderated effects of CVE on
depression (Δ.R2=.14)**, but not on
PTSD symptoms. Presence of mother
did not have a moderating effect for
PTSD or depression.
Ozer, 2005 Metropolitan Public MS
N=73 participating at two data
points
Age range not provided-7th and
8th graders
55% Female
45% Male
18% African American
40% Latino
7% White or European
American
No data on SES
Longitudinal-1 yr
Violence Exp: CREV
PTS and Anxiety: TSCC
Depression: CDI and teacher report of
TRF
Aggression: 6 items from the
perpetration of violence scale developed
by the National Longitudinal Study of
Adolescent Health and teacher report of
TRF
(Teacher report and adolescent report
scored separately)
CVE correlated with self-reported
intrusive (.32)** and anxiety (.28)*
symptoms and aggression (.67)**.
(Regression) CVE predicted in increases
of intrusive (Δ.R2=.12)**, anxiety
(Δ.R2=.09)* symptoms and aggression
(Δ.R2=.37)***. Low mother
helpfulness moderator for CVE and
increase in aggression (Δ.R2=.04)**,
and depressive symptoms (Δ.R2=.07)**.
More helpful mothers moderator for
CVE and decreased depressive
symptoms (Δ.R2 not reported). Less
helpful siblings moderator for CVE and
depressive symptoms (Δ.R2=.05)**. No
relationship with fathers. School
connectedness not a moderator.
Ozer & McDonald, 2006 Metropolitan Public MS
N=71
Median Age 12
55% Female
45% Male
100% Chinese American
No data on SES
Cross-sectional
Violence Exp: CREV
PTSD: TSCC
Depression: CDI and teacher report of
TRF
Perpetration of Violence: 6 items from
National Longitudinal Study of
Adolescent Health Anxiety and teacher
report the TRF
(Teacher report and adolescent report
scored separately)
CVE correlated with self-reported
depressive (.41)**, PTSD (.51)**
symptoms and perpetration of violence
(.60)**.
(Regression) CVE predicted self-
reported depressive (Δ.R2=.08)*, PTSD
symptoms (Δ.R2=.13)**, and
aggression (Δ.R2=.08)*. Together, daily
hassles and CVE was a powerful
predictor of depressive symptoms (total
R2=.27) PTSD symptoms (total R2=.42)
and aggression (total R2=.34). CVE did
not predict teacher report of
anxiety/depressive symptoms.
Ozer & Weinstein, 2004 Metropolitan Public MS
N=349
Median Age 12
48% Female
52% Male
21% African American
39% Asian American
28% Hispanic
No data on SES
Cross-sectional
Violence Exp: CREV
PTSD: TSCC
Depression: CDI and teacher report of
the TRF
Anxiety: Teacher completed the TRF
(Teacher report and adolescent report
scored separately)
CVE correlated with self-reported
depressive (.15)** and PTSD (.29)**
symptoms.
(Regression) CVE predictive of self-
reported depressive (Δ.R2=.01)* and
PTSD symptoms (Δ.R2=.03)**.
Less helpful mother moderator for CVE
and depressive (Δ.R2=.02)* and PTSD
(R2=.01)* symptoms. Less helpful father
moderated for CVE and PTSD
(Δ.R2=.02)*. Less helpful sibling
moderated for CVE and depressive
(Δ.R2=.01)*.
Richards et al., 2004 8 Public ES in Chicago
N=167
Median age 12.5
54% Female
46% Male
100% African American
Median incomes $12,852 to
$37,892
Cross-sectional
Violence Exp: “Things”
Delinquency: SRDS
Aggression and Delinquency: Parent
report of the CBCL
Stress/PTSD: CCDS
Stress/Anxiety/Depression: Parent
Report of CBCL
(When adolescent and parent report not
factored together, parent measure
dropped)
(SEM) (parameter estimates) CVE
related to stress (.49)* and delinquency
(.89)*. CVE partially mediated the
effect of time in risky context (% time
with friends, unmonitored free time,
unstructured leisure time, socialization,
& media use) on stress and delinquency.
CVE partially mediated the effect of
time in protective context (% structured
free time & time with family) on stress
and delinquency.
Rosenthal, 2000 Public 4 year College in NYC
N=455
Median age 18
75% Female
25% Male
50% African American
20% Latino
Median $30,000/yr
Cross-sectional
Violence Exp: SECV
Anger, Anxiety, Depression &
Dissociation: TSI
V and W correlated with anger (.30)*
(.35)*, anxiety (.21)* (.20)*, depression
(.17)* (.13)* and dissociation (.18)*
(.20)* symptoms.
(Regression) Total CVE
(V+W+Overalp) related to anger
(R2=.16)**, anxiety (R2=.06)**,
depression (R2=.04)**, and dissociation
(R2=.05)**.
Rosenthal & Hutton, 2001 Community sample living in
area of NYC public college
N=92
Ages 16–20
50% Female
50% Male
100% African American
55% below $35,000/yr
Cross-sectional
Violence Exp: SECV
Anger, Anxiety, Depression &
Dissociation: TSI
No significant differences in the sample
of non-college students and college
students in the correlations between
total CVE, V and W and psychological
outcomes.
Rosenthal & Wilson, 2003 Public 4 year College in NYC
N=468
Ages 17–19
70% Female
30% Male
34% African American
42% Latino/Hispanic
Median $30,000/yr
Cross-sectional
Violence Exp: Modified Version of the
SECV
Anger, Anxiety, Depression &
Dissociation: TSI
Distress symptoms associated with W
(.22)** V (.19)**, and CVE (V+W)
(.25)**, .
Ruchkin et al., 2007 Public HS data from SAHA
N=1488 at two data points
Mean age 12.4
52.1% Female
47.9% Male
61% African American
23% Hispanic-American
76% on FLP
Longitudinal-2 yr
Violence Exp: Modified SCEVC
PTS: PTSDRI-C
Depression & Anxiety: BASC
Commission of Violence: 6 items from
the SAHA
During Year 1 in females/males, W
correlated with anxiety (.13)(.14)***,
depression (.18)(.17)***, PTS
(.34)(.39)*** and commission of
violence (.36)(.37)***: V with anxiety
(.21)***(.09)*, depression
(.30)(.19)***, PTS (.33)(.36)*** and
commission of violence (.33)(.44)***
During Year 2 in females/males, W
correlated with depression
(.09)*(.12)***, PTS (.24)(.29)***,
commission of violence (.40)(.46)***
and anxiety males only (.09)*: V with
anxiety (.09)*(.22)***, depression
(.19)(.21)***, PTS (.28)(.20)*** and
commission of violence (.29)(.48)***:
(SEM) PTS (Year 2) fully mediated
association of V with anxiety, partially
with depression.
PTS served as a partial mediator for V
and W and commission of violence in
males (not females).
Schwab-Stone et al., 1999 Urban public schools (6th 8th
10th grades) from SAHA
N=2600 participating at two
data points.
Grades 6–8
53% Female
47% Male
52% African American
25% Latino
49% on FLP
Longitudinal-2 yr
Violence Exp: Victimization by
Violence and Witnessing Violence
Questions (Modified from the subscales
of the CECV)
Depression, Anxiety & Antisocial
Behavior: BASC
Willingness to use physical aggression:
4 items from the NASHS
For Initial/Follow-up Sample: CVE
associated with internalizing
(.38)(.40)*** (depression and anxiety)
and externalizing symptoms
(.74)(.75)*** (willingness to use
aggression and antisocial behavior).
(SEM) Grade level moderated
relationship between CVE and
internalizing (younger more likely to
report than older). Strength of
association between CVE and
internalizing and externalizing not
moderated by gender or ethnicity. CVE
in Year 1 associated with internalizing
(just younger (.43)***) and
externalizing (younger (..68)***& older
(.61)***) 2 years later.
Self-Brown et al., 2006 Public MS & HS in mid-size
city
N=121 pairs of adolescents and
parents
Ages 13–16
50% Female
50% Male
97% African American
71%<$20,000/yr
Cross-sectional
Violence Exp: SAVE
PTS & Depression: TSCC
CVE correlated with adolescent
depression (.37)** and PTSD (.41)**.
(Regression) Parental mental health
(PTSD severity score) was a moderator
between adolescent CVE and PTSD, but
not between CVE and depression.
White et al., 1998 Urban public school 6th grade
N=385 participating at two
data points.
Age at T1: 11–14.4
53% Female
47% Male
96% African American or
Black
∼52% on FLP
Longitudinal-6 mos.
Violence Exp: “Things”
Anxiety: RCMAS
(Regression) Females who reported high
CVE at Time 1 reported greater
increases in concentration anxiety at
Time 2. Social Support not a moderator
between CVE and anxiety.
*

p<.05,

**

p<.01,

***

p<.001

(ADIS-C) Anxiety Disorders Interview Schedule (Silverman, 1991; Silverman & Nelles, 1988)

(APS-PTS) Adolescent Psychopathology Scale-Posttraumatic Stress Disorder Subscale (Reynolds, 1998)

(BASC) Behavioral Assessment System for Children (Reynolds & Kamphaus, 1992)

(BFS) Behavioral Frequency Scale (Farrell, Danish, & Howard, 1992)

(CBCL) Child Behavior Checklist (Achenbach, 1991a)

(CCDS) Checklist of Children’s Distress Symptoms (Richters & Martinez, 1990a)

(CDI) Children’s Depressive Inventory (Kovacs, 1979, 1985)

(CICS) Cairns Interpersonal Competence Scale (Cairns, Leung, Gest, & Cairns, 1995)

(CREV) Children’s Report of Exposure to Community Violence (Cooley, Turner, & Beidel, 1995)

(CYDS) Chicago Youth Development Study

(EVQ) Exposure to Violence Questionnaire (Reynolds & Mazza, 1995)

(FSSC-R) Fear Survey Schedule for Children-Revised (Ollendick, 1983)

“My Exposure to Violence Interview” (Buka, Selner-O’Hagan, Kindlon, & Earls, 1997)

(NASHS) National Adolescent Student Health Survey (American School Health Association, Association for the Advancement of Health Education, & Society for Public Health Education, 1991)

(NWS-PTSD Module) National Women’s Study PTSD Module (Kilpatrick, Resnick, Saunders, & Best, 1989)

(PCAP) Predatory Crimes Against Persons (Elliott & Ageton, 1980)

(PTSDRI-C) Posttraumatic Stress Disorder Reaction Index-Child (Pynoos et al., 1987)

(RADS) Reynolds Adolescent Depression Scale (Reynolds, 1986)

(RCMAS) Revised Children’s Manifest Anxiety Scale (Reynolds & Richmond, 1978)

(SAHA) Social and Health Assessment (Weissberg, Voyce, Kasprow, Arthur, & Shriver, 1991) (Schwab-Stone et al., 1999)

(SAVE) Screen for Adolescent Violence Exposure (Hastings & Kelly, 1997)

(SCECV) Survey of Children’s Exposure to Community Violence (Richters & Saltzman, 1990a)

(SECV) Survey of Exposure to Community Violence (Richters & Saltzman, 1990b)

(SCL-90-R) Symptom Checklist 90-Revised (Derogatis, 1994)

(SIQ-J) Suicidal Ideation Questionnaire-Junior (Reynolds, 1987)

(SRDS) Self-Report Delinquency Scale (Elliott, Huizenga, & Ageton, 1985)

(STAIC) State-Trait Anxiety Inventory for Children (Speilberger, 1973)

(“Things”) “Things I Have Seen and Heard” (Richters & Martinez, 1990b)

(TRF) Teacher Report Form (Achenbach, 1991c)

(TSCC) Trauma Symptom Checklist for Children (Briere, 1996)

(TSI) Trauma Symptom Inventory (Briere, 1995)

(WAI) Weinberger Adjustment Inventory (Weinberger, Feldman, Ford, & Chastain, 1987)

(YSR) Youth Self Report (Achenbach, 1991b)

Results

An integrative review of the 26 articles produced seven longitudinal and 19 cross-sectional designs. No papers had a lead investigator who was a nurse and only one nurse scholar was a secondary author among the 26 articles (Davis of McGee et al., 2001). Seventeen studies had samples with greater than 50% African American adolescents and four were predominately Hispanic/Latino populations. Almost all of the studies were conducted with adolescents of disadvantaged socioeconomic status. Eleven different measurements of CVE were used. The time frame for exposure varied, including the prior six months, year, three years, during high school, and over the adolescent’s lifetime. All studies used adolescent self-report measures of CVE and mental health variables. Six studies included parent or teacher report of adolescent behaviors (Gorman-Smith & Tolan, 1998; Ng-Mak et al., 2004; Ozer, 2005; Ozer & McDonald, 2006; Ozer & Weinstein, 2004; Richards et al., 2004). Two studies used experience sampling methods (ESM) to obtain information on daily experiences to supplement self-report data (Hammack, Richards, Luo, Edlynn, & Roy, 2004; Richards et al., 2004).

Parameters of CVE differed across studies. Investigators measured a combination of victimization, witnessing, and/or hearing about violence in the community – either in isolation or together. Of the 24 studies assessing both victimization and witnessing, 11 statistically examined the relationship of victimization and witnessing with adolescent mental health symptoms separately (Cooley-Quille et al., 2001; Duckworth et al., 2000; Foster, et al., 2004; Hammack et al., 2004; Howard et al., 2002; Richards et al., 2004; Rosenthal, 2000; Rosenthal & Hutton, 2001; Rosenthal & Wilson, 2003; Ruchkin, Henrich, Jones, Vermeiren, & Schwab-Stone, 2007; Schwab-Stone et al., 1999). Two assessed witnessing only (Farrell & Bruce, 1997; White et al., 1998). All others assessed CVE as a composite of witnessing and victimization. Three explicitly stated that sexual assault was included in their measurement of CVE (Duckworth et al., 2000; Moses, 1999; Ng-Mak et al., 2004). Many used a modified version of established instruments, with specific modifications, deletions or additions not always explicit. Only one study did not use an established instrument (Moses, 1999). All but three studies used CVE instruments developed in low-income neighborhoods with minority children and adolescents (Gorman-Smith & Tolan, 1998; McCart et al., 2007; Self-Brown et al., 2006).

Twenty-seven different instruments were used to assess depressive symptoms, PTSD symptoms, anxiety, distress, aggression, and suicidal ideation. In all studies, analysis of the relationships between CVE and mental health correlates were based on symptom severity through correlation, regression, and structural equation modeling (SEM). Some reported descriptive statistics of scores indicative of clinical significance, but these were not used in analysis of relationships (Duckworth et al., 2000; Foster et al., 2004; Mazza & Reynolds, 1999; McCart et al., 2007; Overstreet, Dempsey, Graham, & Moely, 1999; Rosenthal & Wilson, 2003; Self-Brown et al., 2006).

Anxiety, Depressive Symptoms, and Suicidal Ideation

Although there was a substantial amount of investigation into anxiety and depressive symptoms as unique symptoms, the discussion of depressive symptoms and anxiety at times overlapped with the terms internalizing symptoms, emotional distress, and anxiety/depression. Studies did not specify that these were synonymous, but acknowledged that the two were related or measured together (Gorman-Smith et al., 1998; Schwab-Stone et al., 1999). Strengths of correlations between CVE and depressive symptoms ranged from r=.11 to r=.51 and for CVE and anxiety from r=.20 to r=.50 (Cooley-Quille et al., 2001; Dempsey, 2002; Foster et al., 2004; Hammack et al., 2004; Mazza & Reynolds; 1999; Moses, 1999; Ozer, 2005; Ozer & McDonald, 2006; Ozer & Weinstein, 2004; Rosenthal, 2000; Ruchkin et al., 2007; Self-Brown et al., 2006).

One study indicated a moderate association between CVE and suicidal ideation for both males and females (Mazza & Reynolds, 1999). Two found no significant relationship between CVE and depressive symptoms (Cooley-Quille et al., 2001; Overstreet et al., 1999); one found no significant relationship between CVE and emotional distress (measured as depression and anxiety) (Farrell & Bruce, 1997); and one found no relationship between CVE and anxiety in males (White et al., 1998). Two reported significant small to moderate correlations with symptoms of anxiety and depression combined as one variable (Gorman-Smith & Tolan, 1998; Schwab-Stone et al., 1999). Overall, a majority indicated a relationship between CVE and depressive symptoms and anxiety.

Regression analyses occasionally revealed a different relationship between CVE and depression from simple correlation. Controlling most often for age and gender, studies specified that CVE accounted for 7–11% of the variance in depressive symptoms and anxiety (Dempsey, 2002; Foster et al., 2004; Overstreet et al., 1999). Even though there was a significant positive moderate correlation between CVE and depressive symptoms, when PTS and suicidal ideation were controlled for, the relationship lost significance (Mazza & Reynolds, 1999). Similarly, CVE and suicidal ideation had a moderate association, but lost significance when depressive symptoms and PTS were controlled for (Mazza & Reynolds, 1999). Controlling for the other mental health variables was not a consistent step in the studies analyzing CVE and depressive symptoms and anxiety, but proved to be an important strategy in determining the strength of variation accounted for by a single variable. In addition, when controlling for previous symptom levels, CVE still accounted for a small amount of the variance in changes of anxiety/depression (Gorman-Smith & Tolan, 1998) and intrusive symptoms (Ozer, 2005).

Three studies used SEM to assess the relationship between CVE and depressive and anxiety symptoms. Schwab-Stone et al. (1999) pointed to the strengths of SEM, in which data on multiple measurements of well-being could be aggregated into latent constructs and measurement error is incorporated to reflect a more accurate picture of the relationship between CVE and mental health symptoms. Indeed, studies using SEM provided new and more refined information about CVE and mental health. Using emotional distress as an inferred latent variable for anxiety and depression, no relationship was found with CVE (Farrell and Bruce, 1997). CVE was associated only with internalizing symptoms (anxiety and depression) in younger adolescents, and was not related to changes in emotional distress (anxiety/depression) over time (Farrell & Bruce, 1997; Schwab-Stone et al., 1999).

Posttraumatic Stress

The relationship of PTSD, the symptom clustering of intrusive recollections, avoidant/numbing symptoms, and hyper-arousal symptoms, and CVE was unique (American Psychiatric Association, 2000). The terms distress, PTS, and PTSD were used to describe some of the symptoms associated with PTSD in this sample. No study used the clinical diagnosis of PTSD. Thus, the term PTS will be used to discuss the findings associated with distress, PTS, and PTSD symptoms.

Positive correlations between PTS and CVE were generally significant with small to moderate effect sizes (r=.19-r=62) (Cooley-Quille et al., 2001; Dempsey, 2002; Duckworth et al., 2000; Foster et al., 2004; Mazza & Reynolds, 1999; McCart et al., 2007; Overstreet & Braun, 2000; Overstreet et al., 1999; Ozer & Weinstein, 2004; Rosenthal & Wilson, 2003; Self-Brown et al., 2006). Regression analysis also supported a relationship, with CVE predicting 3–19% of the variance of PTS (Cooley-Quille et al., 2001; Dempsey, 2002; Duckworth et al., 2000; Foster et al., 2004; Howard et al., 2002; McCart et a., 2007; Ng-Mak et al., 2004; Ozer & Weinstein, 2004). When controlling for depression and suicidal ideation, Mazza and Reynolds (1999) still found CVE to be a predictor of PTS. In regression analysis, victimization and witnessing separately were not as strong predictors of PTS as when they co-existed. Ozer and McDonald (2006) found that daily hassles and CVE were powerful predictors, together accounting for 42% of the variance in PTS. One study used SEM to assess CVE and PTS, indicating a moderate relationship between CVE and the latent variable of stress (Richards et al., 2004).

Aggression

Aggression is defined as hostile or violent behavior or attitudes (Soanes & Stevenson, 2006). Aggression in adolescents was referred to by a variety of terms, including externalizing behavior, delinquency, perpetration of violence, antisocial behavior, hostility, anger, and aggressive behavior. These terms are not interchangeable, but measurements of these variables were used to reflect aggression in adolescents. Thus, the term aggression will be used as an indicator of these terms. Aggression had moderate to strong relationships with CVE, both in correlation and regression analysis. Correlation strength measured r=.14 for a single description of CVE (being beaten up/jumped) (Moses, 1999) and r=.30 to r=.75 for CVE as summed by the instrument (Farrell & Bruce, 1997; Foster et al., 2004; Gorman-Smith & Tolan, 1998; McCart et al., 2007; Ozer, 2005; Ozer & McDonald, 2006; Ruchkin et al., 2007; Schwab-Stone et al., 1999). Overall, these correlations were stronger than those associated with PTS, depressive symptoms, or anxiety.

In regression analysis, CVE accounted for 5–37% of the variance in aggression (Duckworth et al., 2000; Foster et al., 2004; Gorman-Smith & Tolan, 1998; McCart et al., 2007; Ng-Mak et al., 2004; Ozer, 2005; Ozer & McDonald, 2006; Rosenthal, 2000). A strong association between CVE and aggression remained when using SEM, and CVE was predictive of aggression in females (Farrell & Bruce, 1997; Richards et al., 2004; Ruchkin et al., 2007; Schwab-Stone et al., 1999). Ozer and McDonald (2006) found that CVE and daily hassles were powerful predictors of aggression (total R2=.34).

Victimization and Witnessing of CVE

Not all studies distinguished victimization and witnessing of violence. Yet studies that did make this distinction found differential impact of CVE on mental health. Rosenthal (2000) found that victimization and witnessing CVE were independently related to various distress symptoms. Victimization had a slightly stronger association with depression, and witnessing had a slightly stronger association with anger, though both had small to moderate relationships and predicted a small amount of the variance. Overall, there was no consistency in the outcomes of victimization and witnessing. Rosenthal (2000) concluded that in order to assess the full impact of CVE, as well as their overlapping relationship, victimization and witnessing must be considered together.

Some investigators measured and analyzed victimization and witnessing separately and found that they were highly correlated (r=.50) and showed similar relationship to symptoms (both r=.26 with PTSD symptoms) (Ozer & Weinstein, 2004). Even though victimization and witnessing were correlated, Hammack et al. (2004) (r=.60, p<.001) and Howard et al. (2002) (r=.66, p<.000), both analyzed each independently, citing that the two were not mutually exclusive experiences. Findings indicated that mental health symptoms associated with the predictive model of victimization (despondency and lack of belongingness) were different than that of witnessing (intrusive thoughts, vigilance/avoidance, and distraction) (Howard et al., 2002). Other investigators also found differences in these relationships (e.g. Cooley-Quille et al., 2001; Foster et al., 2004). Failure to identify consistent findings associated with victimization and witnessing CVE may be due to the implausibility of finding an adolescent in an urban setting who has not experienced one without the other (Overstreet et al., 1999).

Few studies discussed the impact of specific events of CVE. Moses (1999) focused on six single items, assessing the impact of a major traumatic event, such as being shot at or witnessing violence against family. Witnessing violence against family had small, but significant relationships with depression and aggression; being shot/stabbed was also correlated with aggression. Though data were not collected from a standardized instrument, the unique and specific information from these items helped to identify key characteristics of CVE that may be more detrimental to mental health. Three studies included items on sexual assault in the measurement of CVE, all indicating correlations between CVE and distress or depression (Duckworth et al., 2000; Moses, 1999; Ng-Mak et al., 2004). This specific victimization experience may also have a unique impact on mental health symptomatology.

Moderators to Mental Health Symptoms

A moderator is a variable that “affects the direction and/or strength of the relation between the independent or predictor variable and the dependent or criterion variable” (Baron & Kenny, 1986, p. 1174). Nine studies examined moderators for CVE and mental health symptoms in urban adolescents, including gender (Foster et al., 2004; Schwab-Stone et al., 1999), family relationship characteristics (Gorman-Smith & Tolan, 1998; Hammack et al., 2004; Overstreet et al., 1999; Ozer, 2005; Ozer & Weinstein, 2004; White et al., 1998), school connectedness (Ozer, 2005), parental mental health (Self-Brown et al., 2006), ethnicity, and grade level (Schwab-Stone et al., 1999).

Discrepant results were identified for the moderating role of family support. White et al. (1998) found that family support was not a moderator for CVE and anxiety. Gorman-Smith and Tolan (1998) found that CVE was significantly related to aggression for youth with high family structure and youth with low family cohesion CVE was significantly related with increased depression and anxiety. In other studies, the presence or absence of, and helpfulness of mother, father, and siblings varied in their moderating effects (Overstreet et al., 1999; Ozer, 2005; Ozer & Weinstein, 2004). Specifically, the less helpful/absent mother was a moderator for CVE and depressive, PTSD symptoms, and aggression (Overstreet et al., 1999; Ozer, 2005; Ozer & Weinstein, 2004). Hammack et al. (2004) reported that social support (a self-report measure of social support and measures of time spent with family and maternal closeness) was a protective and stabilizing force for adolescents exposed to CVE. This multidimensional framing of social support may have contributed to the findings. Parental mental health was also found to be a moderator for CVE and PTS (Self-Brown et al., 2006). School connectedness was not found to be a moderator of the effects of CVE (Ozer, 2005; Ozer & Weinstein, 2004).

Mediators to Mental Health Symptoms

A mediator is present when variation in the independent variable accounts for variation in the proposed mediator, variation in the mediator accounts for variation in the dependent variable, and when the first two relationships are controlled for, the significant relationship between the independent and dependent variable loses significance (Baron & Kenny, 1986). Six studies examined mediators of CVE and mental health symptoms including negative coping (Dempsey, 2002), community chaos (Duckworth et al., 2000), PTS symptomatology (Mazza & Reynolds, 1999; Ruchkin et al., 2007), neighborhood safety, and family conflict (Overstreet & Braun, 2000). Community chaos, as a significant mediator for witnessing violence and PTS and problem behaviors, places emphasis on the role of community instability (Duckworth et al., 2000). Overstreet and Braun (2000) found neighborhood safety to be a mediator for CVE and PTS. Even though negative coping acted as a mediator for CVE and PTSD, depression, and anxiety, positive coping did not have the inverse effect (Dempsey, 2002).

Investigating the role of PTS as a mediator for CVE helped to further explain the intricate and complex relationship of CVE and mental health symptoms. Investigating PTS as a mediator indicated that PTS was both an outcome of CVE and also preceded other mental health symptoms such as depression, anxiety, or aggression. This is complex because of similarities that may exist between anxiety, depression, aggression, and PTS (Mazza & Reynolds, 1999). In this review, support was found for the mediating role of PTS. PTS functioned as a mediator to depression for witnessing and victimization of CVE (Mazza & Reynolds, 1999; Ruchkin et al., 2007). Gender differences were identified, where PTS acted as a partial mediator for CVE and commission of violence in males, but not females (Ruchkin et al., 2007). The role of PTS as a mediator has important clinical implications for assessment and care of urban adolescents exposed to CVE. PTS symptoms were not only an outcome of CVE, but also a risk factor for the development of other mental health sequelae in urban adolescents.

Gender Differences in Relationships of CVE and Mental Health Symptoms

Males and females alike are exposed to substantial amounts of violence, but the influence of such exposure varied. The majority of samples were predominately female. Findings indicated that there was little consensus on the differing effects of CVE on mental health for males and females. Some results found that females exposed to CVE were more likely to exhibit anxiety and depressive symptoms than their male counterparts (McGee et al., 2001; Moses, 1999). Anxiety was related to CVE only in females (White et al., 1998), as was violent behavior (Farrell & Bruce, 1997). Other results indicated that anxiety was associated with victimization in males, but not females (Foster et al., 2004). Although males were more distressed by victimization than witnessing, females had no difference in the impact of either victimization or witnessing.

There are likely mediating and moderating effects in gender. Schwab-Stone et al. (1999) found that the strength of the association between CVE and internalizing and externalizing behavior was not moderated by gender. Foster et al. (2004), however, found that gender moderated the relationship between witnessing and depressive symptoms and anxiety, with females who witnessed high levels of CVE having higher symptoms of depression and anxiety. Gender did not necessarily have the same moderating effect for victimization and symptoms. Ruchkin et al. (2007) assessed the mediating role of PTS and the findings suggested that males exposed to CVE and who experienced PTS committed more violence. The effects of the gender variable in mediation and moderation analysis presented conflicting results, indicating that further investigation is needed.

Discussion

Key Findings

Although the research shows discrepant findings about the relationship between CVE and mental health symptoms in urban adolescents, there was clear and convincing evidence of an association between CVE and increased depressive symptoms, anxiety, PTS, and aggression. Aggression and PTS had the strongest relationships with CVE. Depression and anxiety had significant correlations with CVE, but with regression analysis, results often lost significance. Controlling for variables and confounders in regression helped to give a clearer picture of the effect of CVE on mental health. After controlling for the other mental health variables and confounders, PTS and aggression had the strongest relationships with CVE. Potentially, the relationship of CVE and depression/anxiety is more complex with findings changing based on the statistical methods of examining the relationships. The relationship, however, with PTS and aggression seem more stable; regardless of simple correlation or more complex approaches, there is always a relationship. SEM augmented the results with the construction of latent variables and inclusion of measurement error to better describe relationships. Additional analysis beyond correlation is needed to assess the relationships between CVE and mental health symptoms.

The role of mediators and moderators in assessing the mechanisms by which CVE influences mental health symptoms is critically important in understanding the mental health status of urban adolescents. Many studies to date indicate that neighborhood and family characteristics may have had a mediating effect for CVE and mental health. Better understanding these mediating effects is key to future investigative efforts. Clarification of the role of PTS as a mediator and an outcome can be helpful in identifying urban adolescents who might benefit from mental health services. CVE is substantial among male and female urban youth, and consequences of exposure did not appear to have consistent findings in gender differences.

Limitations and Gaps in Knowledge

The lack of consistency in the examination of CVE was a major limitation. The discrepancies stem from different time periods of recall for exposure, modification of instruments, and analysis of CVE as victimization, witnessing, or both. Comparing the results of studies that assess the impact of lifetime CVE versus the past year may not be explicating a true understanding of relationships with mental health symptoms. Even among studies using the same instruments, different time frames were used, possibly causing methodological flaws. Witnessing a shooting in the community two years prior to administration of the instrument would not be captured in an evaluation of CVE in the prior year. Recall of the time frame of CVE may not be consistent. The influence of time in development of mental health symptoms can also be of key importance. Additionally, modifications in instruments, such as deletion of items, were not always specified, making it difficult to effectively compare findings across studies. Furthermore, differences among victimization, witnessing, and total CVE were inconsistent. Not all studies separated witnessing and victimization, creating a complicated comparison across studies, nor was there consensus about the value of distinguishing between the two.

Another methodological issue was the lack of information on proximity and relationship of the adolescent to the victim. Failure to collect data on this information limits the knowledge of how these characteristics affect mental health of urban adolescents. Few instruments measuring CVE were extensive, and those that did include more detailed questions about proximity and relationships did not incorporate these results in the data analysis (Duckworth et al., 2000). For example, there did not appear to be assessment of comparing witnessing a murder as opposed to witnessing a fight. Items used by Moses (1999) provided the most detail for proximity and relationship of victim, but these items was not part of a standardized instrument.

The methods and design of these studies point to another limitation. Self-report data are the mainstay of the evaluation of the relationship between CVE and mental health symptoms in urban adolescents. Although few studies incorporated teacher and parent report of behaviors, findings indicated that these assessments did not have the same findings as self-report (Ng-Mak et al., 2004; Ozer & McDonald, 2006; Ozer & Weinstein, 2004). Adolescents experienced symptoms of depression, anxiety or PTS, but adults were not aware or able to see a display of these emotions. In addition, cross-sectional data were collected in the majority of the studies. With cross-sectional data, there is the possibility that urban adolescents experiencing depressive symptoms or anxiety are more at risk to have CVE. Longitudinal analysis supported a strong argument for the directionality of the relationship between CVE and mental health and also allowed for control of previous levels of symptomatology in analysis (Farrell & Bruce, 1997; Gorman-Smith & Tolan, 1998; Ozer, 2005; Ruchkin et al., 2007; White et al., 1998).

Overall, CVE had a strong correlation with, and was a predictor, for aggression. The assessment of aggression, though, sometimes virtually overlapped with CVE. Acts of perpetration of violence, delinquency, and other aggressive behavior by urban adolescents could be argued as reflective of CVE. For example, items regarding CVE included events such as witnessing someone being shot and for perpetration of violence, items included events such as using a weapon to get something from someone (Ozer & McDonald, 2006). The questions assessing these variables may in fact be overlapping. Indeed, externalizing symptoms all appeared to be related to the mental health and well-being of urban adolescents. Many studies examined both internalizing and externalizing symptoms, indicating that these behaviors were reflective of mental health and psychological functioning (Cooley-Quille et al., 2001; Ozer, 2005; Ozer & McDonald, 2006; Schwab-Stone et al., 1999).

Desensitization, or adaptation and emotional numbing to CVE, has been offered as a possible explanation for results which indicate that CVE does not affect mental health status (Fitzpatrick, 1993; Osofsky, Wewers, Hann, & Fick, 1993). Some findings of this review pointed to desensitization when there was no relationship with CVE and mental health symptoms (Farrell & Bruce, 1997; Moses, 1999; Ng-Mak et al., 2004; White et al., 1998). Ng-Mak et al. (2004) also discussed the presence of pathologic adaptation in a small subset of their sample, in which adolescents did not experience internalizing distress symptoms such as being sad, worrying, or crying, but increased their aggressive behavior. McCart et al. (2007), however, found minimal support for desensitization in their probability sample of urban adolescents. In their regression analyses, however, CVE was not isolated from family violence and sexual assault. Overall, significant relationships between CVE and mental health symptoms indicate that desensitization or pathological adaptation may not be the normative response to CVE. In this review, there was no consensus for desensitization or pathological adaptation as an explanation for non-significant findings. The role of mediators, moderators, and other mental health problems such as PTS, may also help to explain non-significant findings.

Detailed information about relationships and proximity of CVE is lacking. Increased knowledge of the effects of witnessing a violent event in close proximity or against a person in close personal relations with the adolescents could help to determine how these factors affect mental health symptoms. Identification of these aspects may be methodologically difficult, but further exploration is needed. Current instruments that do include these characteristics are often modified or the shortened version is used. In addition, knowledge about victimization characteristics would also help to augment the knowledge.

Conclusions

Implications for Mental Health Nursing

Nursing is virtually absent from this body of science. Yet, nurses working in mental health settings have the opportunity to engage in assessment and care of urban adolescents threatened by CVE. Knowledge about identification of urban adolescents exposed to chronic CVE who experience symptoms of depression, aggression, anxiety, or PTS would be an important contribution to nursing research and practice. Relationships between CVE and mental health symptoms exist for urban adolescents, and mental health nurses can position themselves to become actively engaged in identification and care of this vulnerable population. This is a prime opportunity to explore the understanding of the mechanisms to which CVE affects mental health through education, practice, and research.

Education efforts for psychiatric and mental health nursing should target knowledge about the detrimental effects of violence in the community, as well as interventions that are successful in helping adolescents who are vulnerable to CVE. In clinical practice with urban adolescents, assessment for violence exposure, as well as depression, anxiety, aggression, and PTS should be routine. Assessment of CVE, through mode and proximity, can provide nurses in clinical practice knowledge and opportunities for intervention for potential vulnerabilities to mental health consequences in this population. Given the mediating effects of PTS, early screening, identification, and intervention would be beneficial for improved mental health outcomes. Nursing science must also become involved in this area of research. Nursing inquiry into the understanding of effects of CVE will help to identify what augments healthy development in urban adolescents.

The continued high rates of chronic CVE to which urban adolescents are exposed necessitate assessment of the impact and the mechanisms by which CVE influences mental health. The empirical research conducted with urban adolescents and the relationship between CVE and mental health symptoms have created a strong base from which future studies can be designed. Knowledge of these relationships and continued investigation of mediators and moderators for CVE and mental health symptoms remains an important initiative. There is still a great deal of exploration needed to understand the relationships between CVE and mental health in urban adolescents. Nursing science must be involved in this area of health care for urban adolescents.

Acknowledgements

This research was supported by NIH/NINR (2-T32-NR007100) Research on Vulnerable Woman, Children and Families. This publication was also supported by the cooperative agreement number 5 U49 CE001093 from The Centers for Disease Control and Prevention. Its contents are the sole responsibility of the authors and do not necessarily represent the official views of the Centers for Disease Control and Prevention.

Contributor Information

Catherine C. McDonald, T32 Pre-doctoral Fellow 2007–2008, Research on Vulnerable Women, Children, and Families, Center for Health Disparities, University of Pennsylvania, School of Nursing.

Therese R. Richmond, Associate Professor, University of Pennsylvania, School of Nursing.

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