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. Author manuscript; available in PMC: 2012 Jan 27.
Published in final edited form as: Transplantation. 2011 Jan 27;91(2):176–182. doi: 10.1097/TP.0b013e3181fffff2

Fig. 5.

Fig. 5

Role of HO1 in FD-induced kidney protection against cold ischemic injury: studies using HO1 null mice HO1 null (n=4) and WT type mice (n=6) were injected with FD or saline, and their kidneys were subjected to cold storage in fine suspension. The extent of NAG release (a marker for tubular injury) into supernatant was determined at 24 and 48 h of cold storage (* P<0.05 vs. FD by ANOVA).