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. Author manuscript; available in PMC: 2012 Oct 13.
Published in final edited form as: Neuroscience. 2011 Jul 27;193:54–62. doi: 10.1016/j.neuroscience.2011.07.042

Figure 3.

Figure 3

Slow kinetic eIPSCs were GABAA receptor mediated. In the presence of glutamatergic blockade (20 μM NBQX and 100 μM AP5), sIPSCs were common with subthreshold shocks (artifact marks shock timing, uppermost trace). With constant, suprathreshold shocks, eIPSCs were evoked that were blocked reversibly by either gabazine (3 μM) or TTX (300 nM) and were therefore action potential evoked, GABAA events. Similar protocols for eEPSCs (traces not shown) started with gabazine showing that NBQX blocked the fast kinetic events indicating that they were mediated by non-NMDA receptors. Each panel includes ten trials superimposed. Latency for eIPSC was 2.68 ms. All the traces recorded at VH = −50 mV. All data collected during 0.2 Hz stimulation.