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. 2011 Jun 2;21(6):991. doi: 10.1038/cr.2011.93

Ras1CA overexpression in the posterior silk gland improves silk yield

Li Ma 1, Hanfu Xu 2, Jinqi Zhu 1, Sanyuan Ma 2, Yan Liu 1, Rong-Jing Jiang 1, Qingyou Xia 2, Sheng Li 1
PMCID: PMC3203710

Correction to: Cell Research (2011) 21:991; doi:10.1038/cr.2011.36; published online 15 March 2011

The authors apologize for the mislabeling of Figure 1B, the left label should be Fil-GAL4/3XP3-DsRed2, the right one should be UAS-Ras1CA/3XP3-EGFP. In the legend of Figure 1A, and Materials and Methods, DsRed2 was used instead of DsRed. The correct Figure 1B is as follows.

Figure 1.

Figure 1

The binary GAL4/UAS transgenic silkworm system and 4 different phenotypes. (A) The GAL4 (pBac{Fil-GAL4-3XP3-DsRed2}) and UAS (pBac{UAS-Ras1v12-3XP3-EGFP}) plasmid constructs. (B) The UAS lines are crossed with the GAL4 lines to generate the Fil-GAL4/UAS-Ras1CA lines, in which Ras1CA was specifically overexpressed in the posterior silk gland. The progeny of this crossing shows four different phenotypes in terms of eye color (1) neither DsRed2-positive nor EGFP-positive, wild type, [D(−)E(−)]; (2) only DsRed2-positive, GAL4 lines, [D(+)E(−)]; (3) only EGFP-positive, UAS lines, [D(−)E(+)]; and (4) both DsRed2-positive and EGFP-positive, GAL4/UAS lines, [D(+)E(+)].


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