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. 2012 Jul 4;31(13):3029–3030. doi: 10.1038/emboj.2012.163

The location of splenic NKT cells favours their rapid activation by blood-borne antigen

Patricia Barral, María Dolores Sánchez-Niño, Nico van Rooijen, Vincenzo Cerundolo, Facundo D Batista
PMCID: PMC3395101

Correspondence to: facundo.batista@cancer.org.uk

Correction to: The EMBO Journal (2012) 31, 2378–2390. doi: 10.1038/emboj.2012.87

Since the publication of this paper, it has been brought to the authors' attention that the flow-cytometry profiles in one figure panel (Figure 3A) were incorrectly presented and the correct figure is shown here. However, the conclusions drawn from this figure and from the paper remain unchanged.

Figure 3.

Figure 3

NKT cell activation in response to lipid antigen (A) Intracellular IFN-γ (top) and IL-4 (bottom) staining for splenic NKT cells (TCR-β+αGalCer-CD1d tetramer+B220) 2 h after injection of particulate control lipids (left), GalA-GSL (middle) or αGalCer (right). (BD) Confocal microscopy identification of endogenous NKT cells 2 h after antigen injection. (B) Spleen sections were stained with B220 (cyan), CD169 (green), TCR-β (red) and NK1.1 (blue). Bars, 200 μm. (C, D) Percentages (C) and proportion of cells per area (D) for NKT cells in the RP, MZ, B cell follicles (B) and PALS (T). Data represent 2 independent experiments. P, unpaired two-tailed t-test.

The authors apologize for any inconvenience caused.


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