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The Journal of Biological Chemistry logoLink to The Journal of Biological Chemistry
. 2012 Oct 5;287(41):34495. doi: 10.1074/jbc.A112.340992

Tax protein-induced expression of antiapoptotic Bfl-1 protein contributes to survival of human T-cell leukemia virus type 1 (HTLV-1)-infected T-cells.

Héloïse Macaire, Aurélien Riquet, Vincent Moncollin, Marie-Claude Biémont-Trescol, Madeleine Duc Dodon, Olivier Hermine, Anne-Laure Debaud, Renaud Mahieux, Jean-Michel Mesnard, Marlène Pierre, Louis Gazzolo, Nathalie Bonnefoy, Hélène Valentin
PMCID: PMC3464555

VOLUME 287 (2012) PAGES 21357–21370

PAGE 21360:

Fig. 1B and supplemental Fig. S1B: The lysates from uninfected and HTLV-1-infected T-cell lines were loaded onto independent gels. After transfer, the membranes were cut according to the molecular weight markers to detect different proteins as shown on the figures. After revelation of actin, Bfl-1, Bcl-xL, and Tax for uninfected T-cells and Tax, Bcl-2, Bfl-1, and Bcl-xL for HTLV-1-infected T-cells, membranes were stripped and hybridized with actin and Bcl-2 for uninfected cells and actin for infected cells, respectively. The membranes for HTLV-1-infected cells were vertically spliced as two samples were removed as shown in the figure.

PAGE 21365:

Fig. 6C: We have removed the non-informative central part of the agarose gels. We only show the top and bottom of the gels corresponding to the supershift and the free DNA, respectively.


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