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. 2012 Nov 1;139(21):3973–3985. doi: 10.1242/dev.081596

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© 2012.

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial Share Alike License (http://creativecommons.org/licenses/by-nc-sa/3.0), which permits unrestricted non-commercial use, distribution and reproduction in any medium provided that the original work is properly cited and all further distributions of the work or adaptation are subject to the same Creative Commons License terms.

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Fig. 9. — Drastic reduction in EnMT in Erg^ΔEx4/ΔEx4 during valve morphogenesis. (A-F) Hearts of Erg^ΔEx4/+ (A,C,E) and Erg^ΔEx4/ΔEx4 (B,D,F) mouse embryos at E10.5 stained for β-galactosidase and sagittally sectioned. The AVC and OFT mesenchymal cells that form normally in control embryonic hearts (C,E) are drastically reduced in the mutant (D,F). AVC, atrioventricular canal; OFT, outflow tract; A, atrium; V, ventricle.