Abstract
Unlike leukocytes from normal donors, leukocytes from patients with chronic granulomatous disease do not suffer DNA damage on exposure to the tumor promoter, phorbol myristate acetate.
Full text
PDF

Selected References
These references are in PubMed. This may not be the complete list of references from this article.
- Baehner R. L., Nathan D. G. Leukocyte oxidase: defective activity in chronic granulomatous disease. Science. 1967 Feb 17;155(3764):835–836. doi: 10.1126/science.155.3764.835. [DOI] [PubMed] [Google Scholar]
- Birnboim H. C. DNA strand breakage in human leukocytes exposed to a tumor promoter, phorbol myristate acetate. Science. 1982 Mar 5;215(4537):1247–1249. doi: 10.1126/science.6276978. [DOI] [PubMed] [Google Scholar]
- Birnboim H. C., Jevcak J. J. Fluorometric method for rapid detection of DNA strand breaks in human white blood cells produced by low doses of radiation. Cancer Res. 1981 May;41(5):1889–1892. [PubMed] [Google Scholar]
- Curnutte J. T., Whitten D. M., Babior B. M. Defective superoxide production by granulocytes from patients with chronic granulomatous disease. N Engl J Med. 1974 Mar 14;290(11):593–597. doi: 10.1056/NEJM197403142901104. [DOI] [PubMed] [Google Scholar]
- Hohn D. C., Lehrer R. I. NADPH oxidase deficiency in X-linked chronic granulomatous disease. J Clin Invest. 1975 Apr;55(4):707–713. doi: 10.1172/JCI107980. [DOI] [PMC free article] [PubMed] [Google Scholar]
- Roos D., Weening R. S. Defects in the oxidative killing of microorganisms by phagocytic leukocytes. Ciba Found Symp. 1978 Jun 6;(65):225–262. [PubMed] [Google Scholar]
