Skip to main content
Infection and Immunity logoLink to Infection and Immunity
. 2012 Nov;80(11):4055. doi: 10.1128/IAI.00855-12

IbeA and OmpA of Escherichia coli K1 Exploit Rac1 Activation for Invasion of Human Brain Microvascular Endothelial Cells

Ravi Maruvada 1, Kwang Sik Kim 1
PMCID: PMC3486037

AUTHOR'S CORRECTION

Volume 80, no. 6, p. 2035–2041, 2012. Page 2035, column 2, line 7 from bottom. The following should be inserted: “The ibeA mutant was constructed by the in-frame deletion of ibeA. The phenotype was restored to that of the parent strain by complementation with a plasmid containing ibeA, as well as by Tn7 site-specific insertion of ibeA into the second benign site in the chromosome.”

Page 2036, column 2, line 21: “…the role of Rac1 in E. coli K1 entry of HBMEC is not known” should read “the role of Rac1 in E. coli K1 entry of HBMEC is not well understood. Our findings differ from earlier work (24a) and may be due to the use of different endothelial cells. The source and characteristics of endothelial cells used in the earlier report were not described.”

Page 2038, Figure 2B, 2D and 2F: “VAV2 (WT)” should read “VAV2(CA), or constitutively active VAV2.”

Page 2041: The following reference was omitted.

References

  • 24a. Rudrabhatla RS, Selvaraj SK, Prasadarao NV. 2006. Role of Rac1 in Escherichia coli K1 invasion of human brain microvascular endothelial cells. Microbes Infect. 8:460–469. [DOI] [PMC free article] [PubMed] [Google Scholar]

Articles from Infection and Immunity are provided here courtesy of American Society for Microbiology (ASM)

RESOURCES