ROS-mediated vascular homeostatic control of root-to-shoot soil Na delivery in Arabidopsis

Correspondence to: nicholas.harberd@plants.ox.ac.uk

Correction to: The EMBO Journal (2012) 31, 4359–4370. doi:10.1038/emboj.2012.273

Since the publication of this article, the authors have discovered a small error in the labelling of Figure 6B; ‘sss1-1/atrbohF-F3' should read ‘rss1-1/atrbohF-F3'.

The correct Figure 6 is shown here.

Figure 6.

rss1-1 is a novel mutant AtCesA7 allele. (A) Soil-grown plants (genotypes as indicated) grown under control conditions (Control) and 7 days after salt treatment (Salt). Bar=1 cm. (B) Chlorophyll content of soil-grown plants. Genotypes as indicated (salt treatments were as described in Figure 1A). (C) The mutation causing rss1-1 phenotype. The top panel shows the 2-bp deletion (−CA; highlighted by the red arrow) that interrupts AtCesA7 in rss1-1. Reads from rss1-1 and atrbohF-F3 are aligned against TAIR9. The bottom panel shows the genomic structure of AtCesA7. Blue boxes represent coding sequence, green boxes introns, open blue boxes untranslated regions (UTR). (D) Demonstration that rss-1 is a mutant AtCesA7 allele. atrbohF-F3/AtCesA7^irx3-4 is less sensitive to soil salinity compared to atrbohF-F3 and shows a salt sensitivity similar to that of atrbohF-F3/AtCesA7^rss1-1. Bar=1 cm.

The authors apologize for any inconvenience caused.