Abstract
Mouse 3T3 fibroblasts were found to complement, in simian cell variants semipermissive to simian virus 40, a cold-sensitive defect of an early function, but not a nonconditional defect of viral uncoating. The variant simian cells could rescue simian virus 40 from 3T3 transformants, and this capacity was not temperature dependent.
Full text
PDF
Selected References
These references are in PubMed. This may not be the complete list of references from this article.
- Balint R. F., Linzer D. I., Khoury G., Levine A. J. The expression of SV40 large T-antigen in embryonal carcinoma--SV40 transformed somatic cell hybrids. Virology. 1980 Jan 30;100(2):492–494. doi: 10.1016/0042-6822(80)90540-1. [DOI] [PubMed] [Google Scholar]
- Botchan M., Topp W., Sambrook J. Studies on simian virus 40 excision from cellular chromosomes. Cold Spring Harb Symp Quant Biol. 1979;43(Pt 2):709–719. doi: 10.1101/sqb.1979.043.01.079. [DOI] [PubMed] [Google Scholar]
- Fischer-Fantuzzi L., Vesco C. Cold-sensitive growth of simian virus 40 in semipermissive variants of CV1 cells. J Virol. 1982 Sep;43(3):791–799. doi: 10.1128/jvi.43.3.791-799.1982. [DOI] [PMC free article] [PubMed] [Google Scholar]
- Pontecorvo G. Production of mammalian somatic cell hybrids by means of polyethylene glycol treatment. Somatic Cell Genet. 1975 Oct;1(4):397–400. doi: 10.1007/BF01538671. [DOI] [PubMed] [Google Scholar]