Table 1.
Demographics and clinical data (N=60 episodes of RSE)
| Age; median (range) | 24y | (7m–74y) |
| Children (<18y) | 12 | (20%) |
| Female gender | 30 | (50%) |
| Etiology | ||
| Unknown | 34 | (57%) |
| Acute symptomatic | 20 | (33%) |
| non-anoxic brain injury* | 11 | (18%) |
| post-anoxic encephalopathy | 7 | (12%) |
| systemic etiology* | 2 | (3%) |
| Remote symptomatic | 6 | (10%) |
| Prior history of epilepsy | 9 | (15%) |
| Duration of SE (days); median (range) | 26.5d | (1h–10m) |
| CEEG | 59 | (98%) |
| Time from onset of SE to CEEG (median, range) | <24h | (0–17d) |
| Classification of SE | ||
| Generalized convulsive | 14 | (23%) |
| Tonic-clonic | 5 | (8%) |
| Myoclonic | 6 | (10%) |
| Tonic | 3 | (5%) |
| Generalized nonconvulsive | 3 | (5%) |
| Focal convulsive | 4 | (7%) |
| Epilepsia partialis continua | 2 | (3%) |
| Hemiconvulsive | 2 | (3%) |
| Focal nonconvulsive | 38 | (63%) |
| Status epilepticus of infantile spasms | 1 | (2%) |
Data presented as N (row percentage) unless stated otherwise.
Abbreviations: h: hours; d: days; m: months; y: years. CEEG = continuous EEG monitoring; SE = status epilepticus.
*
Causes of non-anoxic brain injury and systemic etiologies included proven infectious (N=4) or autoimmune (N=2; both anti-NMDA) meningo-encephalitis, subarachnoid hemorrhage (N=2), ischemic stroke (N=2), traumatic brain injury (N=1), sepsis-associated encephalopathy (N=1) and posterior reversible encephalopathy syndrome (N=1).