MEDICAL SCIENCES Correction for “BRCA1 promotes the ubiquitination of PCNA and recruitment of translesion polymerases in response to replication blockade,” by Fen Tian, Shilpy Sharma, Jianqiu Zou, Shiaw-Yih Lin, Bin Wang, Khosrow Rezvani, Hongmin Wang, Jeffrey D. Parvin, Thomas Ludwig, Christine E. Canman, and Dong Zhang, which appeared in issue 33, August 13, 2013, of Proc Natl Acad Sci USA (110:13558–13563; first published July 30, 2013; 10.1073/pnas.1306534110).
The authors note that they omitted a reference to an article by Pathania et al. The complete reference appears below.
Furthermore, the authors note that “It is important to note that the role of BRCA1 in response to UV induced replication stress has also been examined by Livingston and colleagues (41). Both studies observed some overlapping phenotypes in BRCA1 depleted cells (for example, the reduction of RPA foci when treated with UV). However, the two studies also have some discrepancies with respect to PCNA ubiquitination. We speculate that these discrepancies may be due to the knockdown efficiency of BRCA1.”
References
- 41.Pathania S, et al. BRCA1 is required for postreplication repair after UV-induced DNA damage. Mol Cell. 2011;44(2):235–251. doi: 10.1016/j.molcel.2011.09.002. [DOI] [PMC free article] [PubMed] [Google Scholar]