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The Journal of Clinical Investigation logoLink to The Journal of Clinical Investigation
. 2000 Sep 1;106(5):715.

Hypoxia/reoxygenation causes inflammatory response in transgenic sickle mice but not in normal mice

DK Kaul, RP Hebbel
PMCID: PMC381298

J. Clin. Invest. 106:411–420 (2000)

During the final stages of producing this manuscript for publication, an error was introduced in Figure 1. The correct version, accompanied by the legend, appears below. We regret the error and have provided corrected reprints to the corresponding author: Dhananjaya K. Kaul, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, New York 10461, USA

Phone: (718) 430-3702; Fax: (718) 430-8969; E-mail: kaul@aecom.yu.edu.

Figure 1.

Figure 1

Representative fluorographs of cremaster venules in βS mice (top) and their corresponding DHR fluorescence profile analysis (bottom). (a) Normoxic control. (b) After 3 hours of hypoxia and 30 minutes of reoxygenation, the peaks of DHR activity correspond to fluorescence intensity in the vascular endothelial cells. Bar = 10 µm.


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