IMMUNOLOGY. For the article “Diversity of regulatory CD4⁺ T cells controlling distinct organ-specific autoimmune diseases,” by Marie-Alexandra Alyanakian, Sylvaine You, Diane Damotte, Christine Gouarin, Anne Esling, Corinne Garcia, Séverine Havouis, Lucienne Chatenoud, and Jean-François Bach, which appeared in issue 26, December 23, 2003, of Proc. Natl. Acad. Sci. USA (100, 15806–15811; first published December 12, 2003; 10.1073/pnas.2636971100), due to a printer's error, the first bar for the cell population CD25^-CD62L⁺ in Fig. 2 should be open instead of filled. The corrected figure and its legend appear below.

Fig. 2.

Gastritis, colitis, and diabetes in NOD SCID mice reconstituted with spleen cells depleted of distinct regulatory T cell subsets. Incidence of overt diabetes and histological gastritis and colitis in a total of 194 NOD SCID mice reconstituted with various T cell subsets is shown. The major finding was that CD25^-CD62L⁺, CD4⁺CD45RB^high, and CD4⁺CD62L^- T cells were unique in their capacity to trigger almost exclusively one disease, namely gastritis, colitis, or diabetes, respectively. Intermediate patterns, both in terms of incidence and severity (see also Table 1), were observed for the other purified subsets.