Abstract
Racial disparities in pain treatment pose a significant public health and scientific problem. Prior studies demonstrate clinicians and non-clinicians are less perceptive, and suggest less treatment for, the pain of African Americans, relative to European Americans. Here we investigate the effects of explicit/implicit patient race presentation, patient race, and perceiver race on pain perception and response. African American and European American participants rated pain perception, empathy, helping motivation, and treatment suggestion in response to vignettes about patients’ pain. Vignettes were accompanied by a rapid (implicit), or static (explicit) presentation of an African or European American patient’s face. Participants perceived and responded more to European American patients in the implicit prime condition, when the effect of patient race was below the level of conscious regulation. This effect was reversed when patient race was presented explicitly. Additionally, female participants perceived and responded more to the pain of all patients, relative to male participants, and in the implicit prime condition, African American participants were more perceptive and responsive than European Americans to the pain of all patients. Taken together, these results suggest that known disparities in pain treatment may be largely due to automatic (below the level of conscious regulation), rather than deliberate (subject to conscious regulation) biases. These biases were not associated with traditional implicit measures of racial attitudes, suggesting that biases in pain perception and response may be independent of general prejudice.
Perspective
Results suggest racial biases in pain perception and treatment are at least partially due to automatic processes. When the relevance of patient race is made explicit, however, biases are attenuated and even reversed. We also find preliminary evidence that African Americans may be more sensitive to the pain of others than European Americans.
Keywords: racial disparities, pain, empathy, implicit racial bias, explicit racial bias
Introduction
Racial disparities and inequity in pain treatment pose a significant public health and scientific problem. Disparities in people’s response to the pain of others, as well as clinical pain treatment have been well-documented. Compared to the racial majority in America, African Americans are more likely to receive inferior or inadequate pain treatment.2,3,8,11,14,29,47,62,63 Evidence suggests that these disparities may, in part, be related to racial disparities in clinician perception and response to pain.12,14,65 However, the mechanisms underlying these disparities are not well understood.
The subjective nature of pain and the clinical reliance on subjective patient reports for pain assessment may contribute to disparities in clinician response. Several studies have demonstrated that physician pain perception differs from patient pain ratings,36,42,55,60 and can influence decisions about diagnosis and treatment.7 Clinicians’ stereotypes about sociodemographic groups24, 32, 42,66 also affect medical judgments. Importantly, in the absence of objective measures of pain, medical judgments related to pain appear particularly vulnerable to physician bias.5,28
In addition to the influence of stereotypes, disparities in clinician pain treatment may be influenced by cognitive differences in pain perception, empathy, trust, or other intra- and inter-individual factors. While much of the research on disparities in pain treatment has used observational or epidemiological methods, a few recent controlled experiments have demonstrated corresponding racial disparities that favor European Americans in pain perception64, empathy20, and treatment recommendation.20,64 However, other experiments have found no racial bias in pain perception33,35, patient trust33, or treatment recommendation,59,68 and one found a bias favoring African Americans.58 One possible reason for the somewhat inconsistent experimental evidence is the reliance of all of these studies on explicit experimental methods that make the relevance of patient race apparent.
Whereas methods that present race explicitly primarily capture deliberate and consciously held beliefs and values, subtle implicit methods are designed to capture automatic reactions, which may be more reflective of common biases in the culture.23,53 Explicit and implicit measures do not exclusively capture variance due to deliberate and automatic cognitive processing, respectively. Meta-analysis suggests that implicit and explicit measures yield somewhat correlated responses (ρ = .24), but that higher order cognitive processes decrease the relationship between automatic bias and responses to explicit methods of bias assessment.37 Therefore, it is likely that prior explicit assessments of the effects of patient race on pain perception have underestimated the effect of automatic biases.
Experimental examination of automatic effects of race on pain perception and response is important because automatic and deliberate (consciously-held) biases often have differential effects on behavior,18,30 and the most effective interventions to combat automatic and deliberate biases may differ.9,10 Moreover, given the intention of most clinicians to provide equal care, clinician contributions to racial biases in health care likely result from automatic, rather than controlled and deliberate, processes. In the context of these egalitarian values, however, automatic biases may be particularly insidious and result in unintended discrimination and health disparities.17
One way to disentangle the effects of automatic and deliberate mechanisms on racial bias is through priming (testing the effect of very subtle exposure to a stimulus on subsequent behavior). Racial priming (e.g., through the rapid exposure to a Black or White face) has been shown to alter visual perception. For example, studies have shown that people are more likely to detect a weapon within a scrambled image22, or misperceive a tool as a gun50 after exposure to the face of a Black, relative to White, male. Recently, researchers found that physicians implicitly primed with the words black or African before reading about a patient with chest pain responded with decreased perception of cardiac risk and fewer referrals to a specialist than did physicians primed with the words white or Caucasian.56 Interestingly, this effect was only observed when the physicians were under experimentally induced time pressure, and not among physicians who had sufficient time to decide on treatment recommendation. However, implicit racial priming has yet to be applied to the study of racial disparities in pain perception.
Furthermore, experimental tests of the effects of perceiver race on racial biases in pain perception or treatment are largely lacking from the literature (but see relevant studies for independent examination of racial bias within European and African American samples64, and a comparison within a small sample1). The first known study to examine the effect of perceiver race in the context of pain perception included 13 African American participants and 62 European Americans, and found that European Americans perceived greater pain-related negative mood among virtual patients compared to African Americans, suggesting there may be a main effect of perceiver race on pain perception.1 The clinical literature suggests that physician-patient racial congruence can affect the length of and satisfaction with medical encounters,15 however research on the effects on patient health outcomes has revealed mixed results (see meta-analytic review46). Experiments on intergroup empathy for acute pain have demonstrated ingroup biases in physiological4 and neural empathic responses,34,70 suggesting there may be ingroup biases in pain perception as well.
Here we sought to bridge several gaps in the literature. We used a 2(racial prime: explicit vs. implicit) × 2(perceiver race: European American, African American) × 2(patient race: European American, African American) factorial design to examine the effects of priming patient race on pain perception and response in people of one’s same or of a different race. We hypothesized that 1) there would be a main effect of patient race consistent with known disparities in pain, such that European American patients are perceived to be in more pain and elicit a greater response from participants, 2) there would be an interaction between prime and patient race such that racial bias would be smaller in the explicit condition, and 3) participants would reveal an ingroup bias in pain perception and response, perceiving and responding more to the pain of same-race patients. Portions of this research were presented in abstract form at annual meetings of the American Pain Society.43,44
Materials and Methods
Participants
Three-hundred and twenty-four student volunteers, 120 self-identified African Americans (76 female, M = 19.11 years old, SD = 2.59), and 204 self-identified European Americans (103 female, M = 18.99 years old, SD = .99), participated in this study and were either given course credit or compensated $5 for a half hour of their time. This study was approved by the Northwestern University Institutional Review Board, and informed written consent was obtained from each participant prior to the experiment.
Procedure
Participants were told to imagine they were working at the Student Health Center at Northwestern University as part of a work-study job. Participants then read ten case reports, which included patients’ names, patients’ description of their pain symptoms, and a pain rating, presented on a computer screen. Ten racially ambiguous names (i.e. Aaron, Chris, Calvin, Erik, Jason, John, Greg, Mark, Carl, Dennis) were chosen from common American male names (www.ssa.gov/oact/babynames). Each case report included a subjective pain rating made by the patient on a scale from 0–10 (0 = no pain, 10 = worst pain imaginable). Pain complaints included back pain, shoulder pain, neck pain, foot pain, finger pain, headache, and toothache. Pain ratings ranged from 2–9 on a 0–10 scale.
Sample case report
Aaron is a sophomore at NU. He has pain in his lower back. He tells you that he thinks he hurt it lifting a heavy cooler earlier that day. He seems to be otherwise healthy, but tells you on a scale from 0 to 10, he would rate his pain an 8.
Racial priming
Racial priming was used to identify ways in which automatic (below the level of conscious regulation) and deliberate (subject to conscious regulation) racial biases might influence perceptions of, and responses to, pain as well as judgments related to treatment. Participants were randomly assigned to either an implicit or explicit racial prime condition.
In the implicit racial prime condition, case studies were preceded by a facial photograph of either an African American or European American male that was presented for 30ms. This experimental timing has been used in similar racial priming studies13,19, 22, 26, and was chosen based on results from prior studies suggesting that an image presented for 30ms is perceptually detectable (people know they saw something), but unidentifiable (people do not know what they saw).69 The reliability and validity of racial priming methods have been demonstrated across numerous studies and in the context of several outcomes of interest. 23,39
Facial stimuli were adapted from a prior study27, with permission from the authors. Photographs depict young adult males with neutral facial expressions (facial expression was controlled for across racial groups27), Consistent with the implicit priming procedures used in prior studies of automatic racial bias,22 the faces were embedded in a forward and backward mask (i.e., a scrambled image). Masks were presented for 100ms each. In the explicit racial prime condition, the first seven seconds of case report presentations were accompanied by a photograph of either an African American or European American male face (Figure 1).
Figure 1.
Sample Trials. Black bars were not included in the experimental stimuli, but are added here to protect the privacy of volunteers.
Two pseudo-randomized versions of each condition were used to control for potential differences across case studies, such that case studies paired with Black racial primes for half the participants were paired with White racial primes for the other half. Versions were counterbalanced within each participant group (by participant gender and race). This design was chosen as it provides considerable control for various extraneous variables and allows differences to be attributed to patient race as opposed to other factors.
Experimenters
Procedures were facilitated by one of two experimenters: a European American male, or a multi-racial female. Post-hoc analyses controlling for experimenter did not alter reported results.
Measures
Pain perception and response questionnaire
After reading each case study, participants were asked to answer seven questions aimed at targeting the source(s) of racial disparities in pain perception: 1) pain perception: How much pain do you think [patient name] is in?, 2) empathy: How badly do you feel for [patient name]?, 3) helping motivation: How likely would you be to help [patient name] out today?, 4) excused absence Do you think [patient name] should be excused from his exam today and offered a makeup exam?, 5) treatment recommendation: Do you think [patient name] should be given prescription pain medication?, 6) perceived trustworthiness: How trustworthy do you think [patient name] is?, and 7) perceived responsibility: How responsible do you think [patient name] is for his current pain?. Each of these questions was answered on an 11-point Likert-type scale (0 = not at all to 10 = very much). Faces were not present when participants made these responses.
Data reduction
Preliminary analyses revealed a similar pattern of response across, and significant correlation among (Table 1), individual outcome variables. Principal axis factoring with direct oblimin rotation (δ = 0) was chosen to determine the factor structure of the pain perception and response questionnaire. Factor intercorrelation was not restricted. The Kaiser-Meyer-Olkin index of sampling adequacy (KMO = .69) and Bartlett’s test of sphericity (p < .001) suggested factor analysis was appropriate. A scree test suggested either a one- or two-factor solution. A two-factor solution resulted in two correlated subscales (R = .34, p <.001 ), and one item that did not load well onto either factor (perceived responsibility). Given this result, a single six-item composite score (including all questions except perceived responsibility) was created. The six included variables (pain perception, empathy, helping motivation, excused absence, treatment recommendation, and perceived trustworthiness) were z-score transformed and then averaged to form a composite pain perception and response (PPR) score. Separate PPR scores were calculated across patient races (αtotal = .72), in response to African American (AA) patients only (αAA patients = .73), and in response to European American (EA) patients only (αEA patients = .72). Alpha coefficients suggested that the composite score is reliable according to standards in behavioral research, and that the variables are assessing the same latent construct.
Table 1.
Correlation (R) between outcome variables
| Pain | Empathy | Help | Exam | Rx | Trust | |
|---|---|---|---|---|---|---|
| Empathy | .31** | - | - | - | - | - |
| Help | .19** | .64** | - | - | - | - |
| Exam | .26** | .33** | .22** | - | -- | - |
| Rx | .24** | .22** | .13* | .45** | - | - |
| Trust | .11* | .47** | .52** | .13* | .23** | - |
| Responsibility | .12* | .18** | .00 | .12* | .12* | .04 |
p<.05;
p≤.001.
Implicit and explicit measures of racial attitudes
Following the experiment, all participants were asked to complete the Implicit Association Task (IAT31) as a measure of automatic race-based evaluations. The IAT is a computer task designed to assess relatively automatic associations between concepts. Participants in the present study completed an IAT wherein the speed with which they matched African American and European American faces with “good” and “bad” nouns was assessed. The IAT score (D, an effect size for an individual’s responses in the task), represents the extent to which participants tend to more easily (more quickly) associate African Americans with “bad” and European Americans with “good” —i.e., a pro-European American attitudinal bias.
European American participants were additionally asked to complete two scales designed to assess prejudice against African Americans: the Modern Racism Scale (MRS45) and the Motivation to Control Prejudice Scale (MCP21). The MRS is a measure of overt racial attitudes (e.g., Discrimination against Blacks is no longer a problem in the United States.) The MCP assesses motivation to appear non-prejudiced (e.g., It’s important to me that other people not think I’m prejudiced.) This is not a measure of bias per se, but rather a measure of consciously-held motivation to avoid revealing racial biases. Both of these scales are widely used, highly reliable, and well validated.25
Results
A 2 (participant race: AA, EA) × 2 (primed patient race: AA, EA) × 2 (prime: Implicit, Explicit) analysis of variance revealed a significant interaction between prime type and primed patient race, F(1,320) = 11.17, p = .001, η2p = .03 such that participants perceived and responded more to the pain of AA patients than EA patients in the explicit prime condition, but more to EA patients than AA patients in the implicit prime condition (Figure 2). This interaction remained significant when controlling for individual differences in automatic racial attitude bias (IAT, F(1,304) = 10.21, p = .002, η2p = .03). Within group analyses reveal that this interaction is marginal among AA participants, F(1,118) = 3.38, p = .07, η2p = .03 and significant among EA participants, F(1,202) = 9.57, p = .002, η2p = .05. Results among AA participants remain marginal after controlling for automatic racial attitude bias (IAT, F(1,108) = 2.90, p = .09, η2p = .03). Results among EA participants remain significant when automatic racial attitude bias (IAT, F(1,195) = 8.95, p = .003, η2p = .04), motivation to control prejudice (MCP, F(1,189) = 8.67, p = .004, η2p = .04), or overt racial attitude bias (MRS, F(1,190) = 8.81, p = .003, η2p = .04) were included as covariates in the analyses.
Figure 2.
Interaction between prime type and primed patient race. Error bars depict standard error.
When participant sex was entered into the model as a covariate, the interaction between prime type and primed patient race remained significant (F(1,316) = 11.21, p = .001, η2p = .03), and a main effect of participant sex emerged (F(1,316) = 4.35, p = .04, η2p = .01), such that female participants perceived and responded more to the pain of all patients, relative to male participants. We further explore the significant patient race by prime type interaction by examining the results for the explicit and implicit prime conditions separately.
Explicit prime
In the explicit prime condition, as noted previously, there was a significant main effect of primed patient race, F(1,157) = 6.17, p = .01, η2p = .04, such that participants perceived and responded to the pain of AA patients (Mz-score = 0.05, SE = 0.05) more strongly than EA patients (Mz-score = −0.05, SE = 0.05), t(158) = 2.51, p = .01, Cohen’s d = .40, (Figure 3). When participant sex was entered as a covariate into the model, the main effect of primed patient race remained significant (F(1,155) = 5.14, p = .03, η2p = .03). There were no significant direct effects of participant sex. No other main effects or interactions were significant (all ps >.10).
Figure 3.

Pain perception and response (PPR) by prime condition. Error bars depict standard error.
Implicit prime
When patient race was implicitly primed, there was a significant main effect of primed patient race, F(1,163) = 5.00, p = .03, η2p = .03, such that participants perceived and responded to the pain of EA patients (Mz-score = 0.05, SE = 0.05) more strongly than AA patients (Mz-score = −0.04, SE = 0.05), t(164) = 2.55, p = .01, Cohen’s d = .40, (Figure 3). Interestingly, there was also a significant main effect of participant race, F(1,163) = 4.10, p = .05, η2p = .02, such that AA participants were more perceptive of and responsive to pain across all patients (Mz-score = 0.12, SE = 0.07), relative to EA participants (Mz-score = −0.06, SE = 0.06), t(163) = 2.02, p = .05, Cohen’s d = .32, (Figure 3). When participant sex was entered as a covariate into the model, the main effect of primed patient race remained significant (F(1,161) = 6.11, p= .01, η2p = .04). However, the main effect of participant race, controlling for participant sex, became marginally significant (F(1,161) = 3.13, p = .08, η2p = .02). There were no significant direct effects of participant sex. No other main effects or interactions were significant (all ps >.10).
Ingroup biases
No ingroup bias in pain perception and response was found in the group comparison (Figure 3). Individual differences in ingroup bias (IAT, MRS) or concerns about bias (MCP) were not significantly correlated with individual differences in ingroup bias (own race patient > other race patient) in pain perception and response (all ps > .10).
Discussion
Here we demonstrate that implicit and explicit race cues can lead to opposing racial biases in pain perception and response. There is extensive epidemiological and clinical evidence of racial disparities in pain, as well as some experimental evidence that people perceive and respond less to the pain of African Americans, compared to European Americans. The experimental evidence to date is inconsistent, however, with some studies finding a bias favoring European Americans, and other studies finding opposite or no racial biases. The majority of prior studies have employed explicit methods such that participants were aware they were responding, and likely being assessed on their differential responding, to African American and European American patients.
To test our hypothesis that automatic, rather than deliberate, processes are primarily associated with racial biases in pain perception and response, as well as provide a potential explanation for the inconsistencies in prior results; we directly compared explicit and implicit experimental manipulation of patient race. Consistent with our hypotheses, we found that participants tended to perceive and respond more to European American patients than African American patients in the implicit prime condition, when the effect of patient race was presumably below the level of conscious control or regulation. The opposite effect was found within the explicit prime condition, such that participants perceived and responded more to the pain of African American patients than European American patients, when patient race was presented explicitly. We hypothesized that racial bias in the explicit prime condition would be attenuated due to the influence of conscious motivations to respond without prejudice and regulation of bias. However, we found that the preferential bias toward African American patients in the explicit prime condition was not fully explained by individual differences in motivation to control prejudice, nor overt or automatic racial attitudes. Future studies are needed to investigate other motivations to not conform to stereotypes or appear biased that may be more closely related to biases in pain. For example, it is possible that a motivation to compensate for known disparities or injustices that have resulted in unequal suffering by African Americans may contribute to enhanced pain perception and response toward African American patients when race is explicitly manipulated. Taken together, these results suggest that known disparities in pain treatment may be largely due to automatic, rather than deliberate processes. Furthermore, this suggests stereotypes or more specific biases, rather than general racial attitude bias may be responsible for observed race-based differences in pain perception and response.
We also found a main effect of perceiver sex on pain perception and response across, but not within, experimental conditions. When explicit and implicit results are examined together, female participants were more perceptive and responsive to patient pain than male participants. While we did not have specific hypothesis related to perceiver sex, this main effect is consistent with a recent study suggesting women may rate the pain of others as more intense than men.51 While there are few studies on perceiver sex differences in the perception of the pain of other people, and most do not find main effects of perceiver sex on pain perception,1,67 hypotheses can be made based on the empathy literature. Several studies have shown that sex differences in empathy are related to differences motivation, not ability, and are due to empathy-related gender role expectations.38,40 Future studies should control for potential confounding factors, such as gender role-related demand characteristics.
Here we also demonstrate an effect of perceiver race on pain perception such that when conscious regulatory processes are not readily available (i.e. in the implicit prime condition), African American participants tended to be more perceptive and responsive than European Americans to the pain of all patients, irrespective of patient race. Though this effect was partially accounted for by participant sex, the participant race effect remained relatively robust, albeit only marginally significant, even after controlling for sex. This is somewhat contrary to prior results that found European Americans were more perceptive of pain-related negative mood among patients than were than African Americans.1 However, this prior study included only a small sample of African American participants, and only one African American male, and therefore may not have been sufficiently powered to identify racial differences in pain perception. To our knowledge, the present study is the first experiment of racial biases in pain perception and response designed to examine the effects of perceiver race and to recruit a balanced sample of African and European American participants.
Some complementary findings have been reported that lend support to our current results. A large survey employing the Implicit Association Task found the African American physicians did not show general automatic racial biases, where physicians of other races (European/White, Hispanic, and Asian) did reveal culturally congruent racial biases.54 A patient-physician interaction study found that African American physicians display more positive non-verbal communication with African American patients than do European American physicians, though these researchers also found an ingroup bias among African American physicians such that they displayed fewer positive non-verbal behaviors when interacting with European American patients.57 Prior research in non-physician samples has demonstrated that African Americans sometimes display general automatic biases against racial ingroup members,52 particularly under certain circumstances (e.g. when perceived negativity toward one’s group is high),41 but that the content, contributions, and outcomes of these biases may differ from those of European Americans.48 Given the small percentage of African American physicians, and the challenges and discrimination African American physicians may themselves face,49 clinical studies of the effect of perceiver race on disparities in pain perception and treatment are difficult. Nonetheless, future studies should further investigate this interesting effect.
We found no relationship between general automatic racial attitude bias (IAT score) and biases in pain perception and response, suggesting that bias in perception and response to pain is different from more general good vs. bad automatic racial evaluations. Other studies have demonstrated general automatic racial biases among clinicians similar to that of the general population.30,54 However, consistent with the present results, other studies of racial bias in pain perception and response have not found a relationship between traditional measures of general automatic racial evaluations and racial biases in pain perception.33,64 Therefore, biases in pain perception may be more domain and/or stereotype-specific. In other words, people may have specific biases in the domain of pain, such as African Americans are tougher, feel less pain, or are less sensitive to pain than European Americans64,67, that are at least partially independent from their more general tendency to evaluate African Americans less positively overall than European Americans.
Strengths and limitations
This is the first study to directly compare implicit and explicit methods in the study of pain perception biases. Prior studies have largely cued patient race explicitly (either in words, pictures, or videos), and have found mixed results20,33,35,58,59,64,68 The present results suggest that patterns of bias may vary depending on the level at which patient race is processed, and presumably degree to which implicit biases can be consciously regulated. Furthermore, to our knowledge, this is one of the first studies to incorporate a full perceiver race by patient race factorial design. However, future studies using similar designs to investigate automatic and deliberate racial biases in pain perception and response among clinicians (e.g., physicians, nurses and others providing direct care) are still needed. There is some experimental evidence that nurses respond with less bias in pain perception than student samples, perceiving African Americans to be in more pain and need of medical treatment than European American patients54 and reporting equal empathy in response to the pain of African and European American patients.20
In the present study the order of the pain perception and response questions was the same across all vignettes and all participants. Here, we found a similar effect of race on all question responses, and therefore created a composite score of pain perception and response. However, future studies are needed that are designed to disentangle potential separable effects of patient race on pain perception, empathy, and treatment decisions (e.g. controlling for order effects by randomizing the order of questions). Additionally, experimenter and participant demographics were not matched in this study. Though we did not find any effects of experimenter in these analyses, future studies may further explore potential experimenter effects.
Finally, future studies should probe the influence of potential mediators of the relationship between patient race and pain perception and response. For example, the effect of socioeconomic factors, such as education, insurance, and access to health care, on the influence of race on pain perception and treatment may be particularly important to understand when translating these findings within a clinical setting.
Future directions: Toward reducing racial biases in pain perception and treatment
We suggest future studies employ both implicit and explicit measures to examine pain-specific racial biases arising in clinical settings. Both automatic and controlled processes contribute to bias in real-world interactions. Therefore, to understand the source and develop interventions for combating racial disparities in pain, we must assess both types of cognitive processing. While skin-color and other cues to patient race are often readily observable in real-world interactions, people may not consciously examine and regulate the effect of these cues on their reactions and behaviors. Specifically, patient race-relevant cues may trigger clinicians’ consciously held beliefs and automatic associations, which may differentially affect perception, diagnosis, and treatment of pain. Experimental methods, such as implicit racial priming, provide useful tools to examine automatic, unconscious, or un-checked influences of patient race on clinician perception and response. Similar examinations have shed light on racial inequalities in other fields such as law enforcement and criminal justice.6, 22
Future studies are also needed to assess the extent to which racial biases in pain perception and response are due to pain-specific stereotypes and attitudes. The development of pain-specific tools to assess bias may be more appropriate than measures of general racial attitudes when examining racial bias in pain perception and response. Should future studies confirm the influence of pain-specific stereotypes and attitudes on pain perception and treatment, we suggest that interventions targeted at automatic biases may be most effective among a population of clinicians with consciously held egalitarian motivations and goals. Social psychologists have found perspective taking interventions (whereby one imagines the thoughts, feelings, and or experiences of another person)61 and prejudice habit-breaking interventions (whereby participants receive training in, practice, and reflect upon the success of automatic bias reducing strategies in their daily lives)16 can decrease automatic racial biases in behavior. These interventions might be beneficially incorporated into medical school and nursing courses, and implemented in clinical practice. Perspective-taking and habit breaking interventions also lead to increased awareness of and concern about discrimination, inequalities, and injustice which may be of particular value in the context of disparities in pain, given the extent of these disparities29 and the insistence of many clinicians that bias does not affect patient care in their own practices.17 Laboratory and clinical investigations of the effectiveness of these interventions in the context of reducing racial biases in pain perception and treatment are needed.
Acknowledgments
We thank J. Li, E. Kim, M. Schurgin and J. Scimeca for assistance with experimental programming and preparation, and P. Reber, D. Bridge, and B. Cheon for helpful feedback. V.A.M and J.A.R. also wish to acknowledge the intellectual support of Cells to Society (C2S): The Center on Social Disparities and Health and the Society, Biology, and Health Cluster at Northwestern University.
Footnotes
Disclosures
This work was conducted as part of V.A. Mathur’s dissertation and was partially supported by the Northwestern University Society, Biology, and Health Cluster Fellowship (V.A.M.), a research grant from the University Research Grants Committee at Northwestern University (V.A.M.), the National Institutes of Health Ruth L. Kirschstein National Research Service Award T32 NS070201 (V.A.M.), and National Institute of Mental Health grant 1R21NS074017-01A1 (J.Y.C.).
All authors declare that they have no conflicts of interest.
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