Abstract
A man aged 41 years with a clinical diagnosis of cardiomyopathy and hepatic insufficiency was found, on angiocardiography, to have a mass, presumably a thrombus, within the left ventricle. A second angiocardiogram performed five days later showed that the thrombus had disappeared. The mechanism invoked was more probably an increase of spontaneous fibrinolysis as described in cirrhotic patients, perhaps helped by heparin, rather than ejection of the mass from the ventricle with silent embolisation somewhere in the systemic circulation.
Full text
PDFImages in this article
Selected References
These references are in PubMed. This may not be the complete list of references from this article.
- FLETCHER A. P., BIEDERMAN O., MOORE D., ALKJAERSIG N., SHERRY S. ABNORMAL PLASMINOGEN-PLASMIN SYSTEM ACTIVITY (FIBRINOLYSIS) IN PATIENTS WITH HEPATIC CIRRHOSIS: ITS CAUSE AND CONSEQUENCES. J Clin Invest. 1964 Apr;43:681–695. doi: 10.1172/JCI104953. [DOI] [PMC free article] [PubMed] [Google Scholar]
- MILLER R. D., JORDAN R. A., PARKER R. L., EDWARDS J. E. Thrombo-embolism in acute and in healed myocardial infarction. II. Systemic and pulmonary arterial occlusion. Circulation. 1952 Jul;6(1):7–15. doi: 10.1161/01.cir.6.1.7. [DOI] [PubMed] [Google Scholar]
- Meltzer R. S., Guthaner D., Rakowski H., Popp R. L., Martin R. P. Diagnosis of left ventricular thrombi by two-dimensional echocardiography. Br Heart J. 1979 Sep;42(3):261–265. doi: 10.1136/hrt.42.3.261. [DOI] [PMC free article] [PubMed] [Google Scholar]
- Perrin A., Loire R., Dupont J. C. Etude anatomique des myocardiopathies d'apparence primitive. Statistique française portant sur 134 observations. Arch Mal Coeur Vaiss. 1972 Jan;65(1):65–73. [PubMed] [Google Scholar]
- Pises P., Bick R., Siegel B. Hyperfibrinolysis in cirrhosis. Am J Gastroenterol. 1973 Sep;60(3):280–289. [PubMed] [Google Scholar]