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. 2015 Dec 29;7(6):6693–6710. doi: 10.18632/oncotarget.6793

Figure 6. Contribution of MYC to the growth defect in HIF-1α/ANXA1-deficient cells.

Figure 6

(A) Representative immunoblot analysis of MYC and β-catenin in control (SCR), HIF-1α-deficient (HIF-), ANXA1-deficient (ANXA-) and double knock-down (HIF-/ANXA-) AGS cells. YY-1 served as loading control. (B) Re-expression of MYC was achieved by transduction with retroviruses containing human MYC and anchorage-dependent proliferation was analyzed 14 days post transduction. Results shown are representative of three independent experiments and values represent the mean ± SEM of triplicate determinations (***P < 0.001).