The authors would like to draw the reader's attention to errors in the following article:
Echizen K, Hirose O, Maeda Y, Oshima M. Inflammation in gastric cancer: Interplay of the COX‐2/prostaglandin E2 and Toll‐like receptor/MyD88 pathways. Cancer Sci 2016; 107: 391–397.
On Page 4:
Interleukin‐11 signaling by the gp130 receptor promotes gastric tumorigenesis through Stat3 activation.(23,52)
The text was corrected and should have read:
Interleukin‐11 signaling by the gp130 receptor promotes gastric tumorigenesis through Stat3 activation.(52)
In the Reference list section, new reference 36 was inserted and reference 51 was deleted. Hence, references 36 to 50 were renumbered as 37 to 51. The correct reference entries from 36 to 51 are shown below:
The authors apologize for these errors and any confusion it may have caused.
References
- 36. Hayakawa Y, Ariyama H, Stancikova J, et al. Mist1 expressing gastric stem cells maintain the normal and neoplastic gastric epithelium and are supported by a perivascular stem cell niche. Cancer Cell 2015; 28: 800–14. [DOI] [PMC free article] [PubMed] [Google Scholar]
- 37. Kulbe H, Chakravarty P, Leinster DA, et al. A dynamic cytokine network in the human ovarian cancer microenvironment. Cancer Res 2012; 72: 66–75. [DOI] [PMC free article] [PubMed] [Google Scholar]
- 38. Oshima H, Ishikawa T, Yoshida GJ, et al. TNF‐α/TNFR1 signaling promotes gastric tumorigenesis through induction of Noxo1 and Gna14 in tumor cells. Oncogene 2014; 33: 3820–9. [DOI] [PubMed] [Google Scholar]
- 39. Block K, Gorin Y. Aiding and abetting roles of NOX oxidases in cellular transformation. Nat Rev Cancer 2012; 12: 627–37. [DOI] [PMC free article] [PubMed] [Google Scholar]
- 40. Laurent E, McCoy JW 3rd, Macina RA, et al. Nox1 is over‐expressed in human colon cancers and correlates with activating mutations in K‐Ras. Int J Cancer 2008; 123: 100–7. [DOI] [PMC free article] [PubMed] [Google Scholar]
- 41. Kodama R, Kato M, Furuta S, et al. ROS‐generating oxidases Nox1 and Nox4 contribute to oncogenic Ras‐induced premature senescence. Genes Cells 2013; 18: 32–41. [DOI] [PubMed] [Google Scholar]
- 42. Myant KB, Cammareri P, McGhee EJ, et al. ROS production and NF‐ƘB activation triggered by RAC1 facillitate WNT‐driven intestinal stem cell proliferation and colorectal cancer initiation. Cell Stem Cell 2013; 12: 761–73. [DOI] [PMC free article] [PubMed] [Google Scholar]
- 43. Ishimoto T, Nagano O, Yae T, et al. CD44 variant regulates redox status in cancer cells by stabilizing the xCT subunit of system xc(‐) and thereby promotes tumor growth. Cancer Cell 2011; 19: 387–400. [DOI] [PubMed] [Google Scholar]
- 44. Gorrini C, Harris IS, Mak TW. Modulation of oxidative stress as an anticancer stratedy. Nat Rev Drug Discov 2013; 12: 931–47. [DOI] [PubMed] [Google Scholar]
- 45. Parkin DM. The global health burden of infection‐associated cancers in the year 2002. Int J Cancer 2006; 118: 3030–44. [DOI] [PubMed] [Google Scholar]
- 46. de Martel C, Ferlay J, Franceschi S, et al. Global burden of cancers attributable to infections in 2008: a review and synthetic analysis. Lancet Oncol 2012; 13: 607–15. [DOI] [PubMed] [Google Scholar]
- 47. Hatakeyama M. Helicobacter pylori CagA and gastric cancer: a paradigm for hit‐and‐run carcinogenesis. Cell Host Microbe 2014; 15: 306–16. [DOI] [PubMed] [Google Scholar]
- 48. Pachathundikandi SK, Lind J, Tegtmeyer N, et al. Interplay of the gastric pathogen Helicobacter pylori with Toll‐like receptors. Biomed Res Int 2015; 2015: 192420. [DOI] [PMC free article] [PubMed] [Google Scholar]
- 49. Wang K, Karin M. Tumor‐elicited inflammation and colorectal cancer. Adv Cancer Res 2015; 128: 173–96. [DOI] [PubMed] [Google Scholar]
- 50. Rakoff‐Nahoum S, Medzhitov R. Toll‐like receptors and cancer. Nat Rev Cancer 2009; 9: 57–63. [DOI] [PubMed] [Google Scholar]
- 51. Hanada K, Uchida T, Tsukamoto Y, et al. Helicobacter pylori infection introduces DNA double‐strand breaks in host cells. Infect Immun 2014; 82: 4182–9. [DOI] [PMC free article] [PubMed] [Google Scholar]