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. 2016 Aug 19;291(34):17535. doi: 10.1074/jbc.A109.095208

Histone deacetylase inhibitors activate NF-κB in human leukemia cells through an ATM/NEMO-related pathway.

Roberto R Rosato, Sarah S Kolla, Stefanie K Hock, Jorge A Almenara, Ankita Patel, Sanjay Amin, Peter Atadja, Paul B Fisher, Paul Dent, Steven Grant
PMCID: PMC5016150  PMID: 27543593

VOLUME 285 (2010) PAGES 10064–10077

This article has been withdrawn by the authors. The image used to represent U937 cells treated without TBAP for 4 h in Fig. 4A was reused to represent U937/3.1EV cells treated with LBH for 4 h in Fig. 7B. In Fig. 4C, lanes 1 and 2 of the left pIKK-β/α panel were duplicated, lanes 1 and 3 of the right pIKK-β/α panel were duplicated, lanes 4 and 5 of the left IKK-β/α panel were duplicated, and lanes 2–5 of the left actin panel were duplicated in lanes 2–5 of the right actin panel. The actin immunoblot in Fig. 6A was reused as the actin immunoblot in Fig. 7A. Part of the actin panels in Fig. 4C was reused as actin in Fig. 6B. In Fig. 6B, lanes 1 and 2 of the left actin panel were duplicated in lanes 2 and 3 of the right actin panel. In supplemental Fig. 1A, lane 3 of the actin immunoblot from Jurkat cells was reused as lane 1 of the actin immunoblot from HL-60 cells. In supplemental Fig. 3A, lanes 1 and 2 of the TRAF2 immunoblot were duplicated. The actin immunoblot from supplemental Fig. 4B was reused as the actin immunoblot in supplemental Fig. 5B.


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