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. 2016 Apr 7;7(24):35874–35893. doi: 10.18632/oncotarget.8624

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Copyright: © 2016 Huang et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A) Procedure for fractionation of EtOAc extracts from A. marina leaves; (B) Antiproliferative effects of F2-5, F3-2-9, and F3-2-10 in AU565, BT483, HepG2, and Huh7 cancer cells were determined using MTT assays. (C) Comparisons of antiproliferative effects of F2-5, F3-2-9, luteolin, quercetin, apigenin, kaempferol, and chrysin in AU565 and BT483 breast cancer cells; (D) Comparisons of antiproliferative effects of F2-5, F3-2-9, paclitaxel, tamoxifen, tdoxorubicin, and rapamycin on AU565 and BT483 breast cancer cells. All values are expressed as means ± standard errors of the mean (SE) and differences were identified using t-tests; *p < 0.05, **p < 0.01, ***p < 0.001.

(A) Procedure for fractionation of EtOAc extracts from A. marina leaves; (B) Antiproliferative effects of F2-5, F3-2-9, and F3-2-10 in AU565, BT483, HepG2, and Huh7 cancer cells were determined using MTT assays. (C) Comparisons of antiproliferative effects of F2-5, F3-2-9, luteolin, quercetin, apigenin, kaempferol, and chrysin in AU565 and BT483 breast cancer cells; (D) Comparisons of antiproliferative effects of F2-5, F3-2-9, paclitaxel, tamoxifen, tdoxorubicin, and rapamycin on AU565 and BT483 breast cancer cells. All values are expressed as means ± standard errors of the mean (SE) and differences were identified using t-tests; *p < 0.05, **p < 0.01, ***p < 0.001.