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. 2016 Dec 20;13:88. doi: 10.1186/s12977-016-0319-0

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© The Author(s) 2016

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Ruxolitinib does not alter apoptosis threshold or CD69 expression in the presence of ingenol-3,20-dibenzoate. Ruxolitinib (100 nM) did not significantly alter the percentage of resting CD4⁺ T cells expressing activated caspase 3 (a) or expression of CD69 (represented as the mean florescence intensity or MFI with the total percentage of cells in each culture condition expressing CD69 within each bar; b) in the presence of ingenol-3,20-dibenzoate (ingenol DB). Mean values and standard deviation are shown for four independent experiments using resting CD4⁺ T cells from aviremic ART-treated participants (n = 4)