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. 2017 Feb 6;7:41465. doi: 10.1038/srep41465

Corrigendum: Modulation of AMPA receptor mediated current by nicotinic acetylcholine receptor in layer I neurons of rat prefrontal cortex

Bo Tang, Dong Luo, Jie Yang, Xiao-Yan Xu, Bing-Lin Zhu, Xue-Feng Wang, Zhen Yan, Guo-Jun Chen
PMCID: PMC5293000  PMID: 28165451

Scientific Reports 5: Article number: 14099 10.1038/srep14099; published online: September 15 2015; updated: February 06 2017

The authors of Article made an error in preparation of the final version of Figure 2 and inadvertently used panel 2C as both panel 2Ab and panel 2C. A revised version of Figure 2 with the correct panel 2Ab is published below. Other panels of this figure are unchanged.

Figure 2. Nicotine effect on AMPA current and AMPA/NMDA ratio are dependent on α7-nAChR and intracellular calcium but not NMDA receptor.

Figure 2

(A) Representative traces of AMPA and NMDA currents before (ctl) and after (nic) nicotine in slices preincubated with DHβE (1 μM, top) or MLA (10 nM, bottom); (B) Bar plot summary of nicotine effect on AMPA/NMDA ratio in DHβE, MLA and control (ctl). **P < 0.001 (nicotine vs. control, paired Student’s t-test, n = 6). (C) Representative traces of AMPA and NMDA currents before (ctl) and after (nic) nicotine treatment with intracellular infusion of BAPTA (10 mM) are shown on the top; histograms and bar plots of AMPA/NMDA ratio are shown on the bottom (P > 0.05, paired Student’s t-test, n = 5). (D) Representative traces (top) and histograms and dot plots (bottom) of AMPA currents before (ctl), after APV (apv, 50 μM) and APV with nicotine (apv +nic). P = 0.368, apv vs. control; *P = 0.009, apv vs. nic +apv; P = 0.013, ctl vs. nic +apv (paired Student’s t-test, n = 6).

This correction does not affect the conclusions of the Article. The authors apologize for the error and any inconvenience caused by it.


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