Skip to main content
Scientific Reports logoLink to Scientific Reports
. 2017 Feb 9;7:41900. doi: 10.1038/srep41900

Corrigendum: ROS-mediated iron overload injures the hematopoiesis of bone marrow by damaging hematopoietic stem/progenitor cells in mice

Xiao Chai, Deguan Li, Xiaoli Cao, Yuchen Zhang, Juan Mu, Wenyi Lu, Xia Xiao, Chengcheng Li, Juanxia Meng, Jie Chen, Qing Li, Jishi Wang, Aimin Meng, Mingfeng Zhao
PMCID: PMC5299957  PMID: 28181509

Scientific Reports 5: Article number: 10181110.1038/srep10181; published online: May 13 2015; updated: February 09 2017

The iron-overload mouse model used in this study was previously reported by the authors in references 1 and 2, which are not cited in the Article.

The data supporting the mouse model is presented in Figure 1 of the Article. The data presented in in Figure 1b appear in reference 1 as Figure 1b; and the data presented in Figure 1c appear in reference 1 as Figure 1c and in reference 2 as Figure 1.

Therefore the legend of Figure 1 should read:

Figure 1. The establishment of an iron overload mouse model. (a) Iron overload induced labile iron pool level (LIP) of bone marrow mononuclear cells (BMMNCs) in a time- and dose-dependent manner showing the mean fluorescent intensity (MFI) of calcein. (N = 3, ***P < 0.001 vs. CTL). (b) The hepatic, splenetic and bone marrow (BM) iron deposits were exposed to hematoxylin-eosin staining (x400). Republished from [1]. (c) BM cells were subjected to Perl’s iron staining (x1,000). Republished from [1], and from [2] with permission.

The authors apologize for the omission of this information in the original Article.

References

  1. Zhang Y. et al. Effects of Iron Overload on the Bone Marrow Microenvironment in Mice. PLoS ONE 10(3), e0120219, doi: 10.1371/journal.pone.0120219 (2015). [DOI] [PMC free article] [PubMed] [Google Scholar]
  2. Chai X. et al. Effects and mechanism of iron overload on hematopoiesis in mice with bone marrow injury. Chin. J. Hematol. 35(11), 1000–4 doi: 10.3760/cma.j.issn.0253-2727.2014.11.011 (2014). [DOI] [PubMed] [Google Scholar]

Articles from Scientific Reports are provided here courtesy of Nature Publishing Group

RESOURCES