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. Author manuscript; available in PMC: 2018 Mar 15.
Published in final edited form as: J Affect Disord. 2017 Jan 5;211:83–91. doi: 10.1016/j.jad.2017.01.011

Perceived Emotional Social Support in Bereaved Spouses Mediates the Relationship between Anxiety and Depression

Nicholas C Jacobson 1,1, Kayla A Lord 1, Michelle G Newman 1
PMCID: PMC5304338  NIHMSID: NIHMS844306  PMID: 28103522

Abstract

Background

Prior research has shown that anxiety symptoms predict later depression symptoms following bereavement. Nevertheless, no research has investigated mechanisms of the temporal relationship between anxiety and later depressive symptoms or examined the impact of depressive symptoms on later anxiety symptoms following bereavement.

Methods

The current study examined perceived emotional social support as a possible mediator between anxiety and depressive symptoms in a bereaved sample of older adults (N = 250). Anxiety and depressive symptoms were measured at Wave 1 (immediately after bereavement), social support was measured at Wave 2 (18 months after bereavement), and anxiety and depressive symptoms were also measured at Wave 3 (48 months after bereavement).

Results

Using Bayesian structural equation models, when controlling for baseline depression, anxiety symptoms significantly positively predicted depressive symptoms 48 months later, Further, perceived emotional social support significantly mediated the relationship between anxiety symptoms and later depressive symptoms, such that anxiety symptoms significantly negatively predicted later emotional social support, and emotional social support significantly negatively predicted later depressive symptoms. Also, when controlling for baseline anxiety, depressive symptoms positively predicted anxiety symptoms 48 months later. However, low emotional social support failed to mediate this relationship.

Conclusions

Low perceived emotional social support may be a mechanism by which anxiety symptoms predict depressive symptoms 48 months later for bereaved individuals.


Bereavement and spousal loss is especially common in later life with 24.7% of adults ages 65 and older being widowed [1]. One study found that over a course of 2.5 years, 8.1% of older adults reported losing a spouse [2]. Bereavement also was associated with both anxiety [39] and depressive symptoms [7; 914]. Additionally in those experiencing bereavement, higher anxiety was associated with higher perceived loss of control [15], lower energy [16], increased suicidal ideation [17], increased risk of a heart attack [17], increased risk of stomach problems [17], poorer health [16], and greater difficulty sleeping [16] compared to those with lower anxiety. Similarly, in those experiencing bereavement, higher depression was associated with higher blood pressure [17], poorer health [17], greater cognitive impairment [18] and poorer coping [19] compared to those with lower depression. Given the wide-ranging influence of anxiety and depressive symptoms in those who have experienced loss, it is important to understand the longitudinal relationship between anxiety and depressive symptoms following bereavement as well as potential mediators of this relationship.

In evidence on the longitudinal relationship between anxiety and depression outside the context of bereavement, a meta-analysis examining 29 studies and over 20,000 people, showed that anxiety symptoms positively predicted later depressive symptoms with an estimated longitudinal association of r = 0.34 [20; 21]. Likewise, anxiety symptoms predicted depressive symptoms across many time scales, ranging from hours to years [2228]. Similarly, in a meta-analysis of the broader literature including approximately 8,000 persons, depressive symptoms predicted later anxiety symptoms with an average longitudinal association of r = 0.35 [20; 29].

Nonetheless, literature on the longitudinal relationship between anxiety and depression in the context of bereavement is much more scarce. Following bereavement, two studies found that anxiety symptoms predicted later depressive symptoms [5; 16]. At the same time, the relationship between depression and later anxiety has not been examined within this context. Thus, despite robust bi-directional associations between anxiety and depression in younger populations, no studies have actually tested whether a bi-directional relationship exists between anxiety and depression in older adult populations. In addition, no prior research has examined the mechanism of this relationship in the context of bereavement or in an elderly population. Studying such mechanisms within older adults will become increasingly important as, by the year 2033, those who are 65-years and older will outnumber those 18 and younger in the United States [30].

There is reason to focus on perceived emotional social support as a possible mediator of this relationship. Following widowhood, the feeling of anxiety and dread about other bad things happening in the future is theorized to lead many either to emotionally and socially withdraw to conserve one’s energy for personal coping [31], or to engage in behaviors that might push others away. The impact of such withdrawal or negative behaviors may be greater in aging widows/widowers, as older adulthood (even outside the context of bereavement) is a time frequently associated with decreasing social support, due to increasing physical barriers and loss in one’s social circles [32]. Thus, bereavement in older adulthood is likely to be a time of considerable anxiety, dread, and tension, and these feelings are likely to lead to greater withdrawal, poor emotional disclosure, and unaffiliative behaviors.

A lack of emotional support following expressions of anxiety may be particularly impactful among older adults. Low social support in this population may contribute to feelings of loneliness, isolation [21; 33; 34], and emotional isolation [21]. By not having sufficient support and outlets to cope with and process the loss, social and emotional isolation may be a primary mechanism between bereavement anxiety and later depression [35]. In sum, high anxiety following widowhood is theorized to lead to friends’ departure, low emotional support, isolation, and ultimately depression.

Some evidence for this theory comes from nonbeareaved younger samples. Higher anxiety symptoms were associated with more difficulty in the social domain [including difficulty making friendships; 36; 37] and more difficulty with emotional self-disclosure [38; 39]. Chronic worrying also has been associated with impacting significant others in unaffiliative ways [40]. In longitudinal studies, anxiety symptoms unidirectionally predicted later low social support, and such lack of social support did not increase subsequent anxiety symptoms [41; 42]. In addition, lower levels of perceived emotional social support predicted higher depressive symptoms longitudinally [10; 12; 43; 44] following negative life events. There is also evidence that perceived emotional social support may be a mechanism by which anxiety leads to depression. One prior study found that perceptions of intimacy in close and group relationships mediated the longitudinal relationship between anxiety and later depressive symptoms [28]. In another study, interpersonal dysfunction mediated the relationship between generalized anxiety disorder and depressive symptoms [45].

Additional evidence comes from cross-sectional data using bereavement samples. For example, anxiety symptoms were associated with low emotional social support in persons who had experienced a stillbirth [which has been noted as a form of bereavement; 46; 47] and in bereaving spouses [48]. In addition, in observations of widows discussing their emotional reactions to the loss of their spouses, those perceived as less well-adjusted evoked greater frustration and less compassion from onlookers, compared to those who were perceived as better adjusted [49]. In turn, deficits in support networks, loneliness, and the amount of emotional support received from one’s friendships have also been cross-sectionally linked to depression in older adults experiencing bereavement [50; 51]. Further, among the elderly experiencing bereavement, a strong relationship has been found between lacking help in making decisions and feeling more blue or depressed [52]. Thus, persons experiencing and expressing their anxiety may elicit low compassion and a lack of emotional social support from others, leading to fewer social resources, lower emotional support, less helping making decisions and ultimately depression.

The goal of the current study was to examine perceived emotional social support as a mechanism of the relationship between post-bereavement earlier anxiety and later depressive symptoms utilizing a longitudinal design in an older adult sample. We also tested whether the relationship between anxiety and depression was unidirectional or bidirectional following bereavement in older adults. Face-to-face interviews were conducted 6-months (Wave 1), 18-months (Wave 2), and 48-months (Wave 3) after a spouse’s death. A body of literature regarding bereavement trajectories supports this timing of measurement points. The precedent in spousal bereavement literature is to assess symptoms post bereavement at 3–7 months, then between 12–18 months, and at multiple time points in years with a particular interest in the first five years after bereavement [5360]. Some research shows that bereavement symptoms are still present decades after the loss [61]. The interviews consisted of a large set of questions including validated anxiety and depression scales. Social support was defined using two highly correlated items. Based on previous research, we hypothesized that: (1) anxiety symptoms (at Wave 1) would positively predict depressive symptoms (Wave 3) three and half years later while controlling for baseline depressive symptoms (Wave 1), (2) depressive symptoms (Wave 1) would positively predict anxiety symptoms (Wave 3) three and a half years later while controlling for baseline anxiety symptoms (Wave 1), (3) emotional social support (Wave 2) would mediate the relationship between anxiety symptoms (Wave 1) and later depressive symptoms (Wave 3), such that anxiety symptoms would negatively predict emotional social support, and emotional social support would negatively predict depressive symptoms, and (4) emotional social support (Wave 2) would not significantly mediate the relationship between depression (Wave 1) and later anxiety (Wave 3).

Method

Participants

Participants (N = 250) were collected through the Changing Lives of Older Couples (CLOC): A Study of Spousal Bereavement in the Detroit Area public use data set [62]. Data collection took place in three waves between 1987 and 1993. Individuals were identified through a two-stage area probability sample [62]. To be eligible for the study, participants needed to speak English, be at least 65 years old, and married at the time of initial contact. Following initial contact, death records of the state of Michigan were monitored, and participants were contacted 6-, 18-, and 48-months following the loss of their spouse [62]. Data were collected through interviews. Wave 1 interviews were conducted 6-months after the loss of a spouse (N = 250; 86% female, M age = 70.11, 84.4% Caucasian, 15.6% African American). Wave 2 interviews were conducted 18-months post-bereavement (N = 210), and the third and final wave of data was collected 48-months post-bereavement (N = 106).

Measures

Anxiety Subscale of the Symptom Checklist-90 Revised

Anxiety symptoms were measured during each wave with the 10-item anxiety subscale of the Symptom Checklist-90 Revised (SCL-90-R) [63], which is a self-report questionnaire designed to measure anxiety symptoms in the general population. See Table 1 for items. The 10-item anxiety subscale has high convergent validity with the Minnesota Multiphasic Personality Inventory (MMPI) anxiety subscale [64] and the Beck Anxiety Inventory (BAI) [65]. Furthermore, high levels of invariance across gender have been noted [66], and high construct validity has been found as well [67]. In the current sample, the internal consistency of this subscale was high (α = .84) [62]. Responses were measured on a five point Likert scale from 1 (not at all) to 5 (extremely).

Table 1.

Anxiety, Depression, and Social Support Scales

Measure Item Scale
SCL-90-R [How much have you been bothered by]
nervousness or shakiness?
Not at all 1—5 extremely
…trembling? Not at all 1—5 extremely
…feeling suddenly scared for no reason? Not at all 1—5 extremely
…feeling fearful? Not at all 1—5 extremely
…heart pounding or racing? Not at all 1—5 extremely
…feeling tense and keyed up in the past seven
days?
Not at all 1—5 extremely
…spells of terror or panic? Not at all 1—5 extremely
…feeling so restless you couldn’t sit still? Not at all 1—5 extremely
…feeling that something bad is going to happen
to you?
Not at all 1—5 extremely
…thoughts and images of a frightening nature? Not at all 1—5 extremely
CES-D I felt depressed. Hardly ever 1—3 most of the time
I felt that everything I did was an effort. Hardly ever 1—3 most of the time
My sleep was restless. Hardly ever 1—3 most of the time
I was not happy. Hardly ever 1—3 most of the time
I felt lonely. Hardly ever 1—3 most of the time
People were unfriendly. Hardly ever 1—3 most of the time
I did not enjoy life. Hardly ever 1—3 most of the time
I did not feel like eating. My appetite was poor. Hardly ever 1—3 most of the time
I felt sad Hardly ever 1—3 most of the time
I felt that people disliked me. Hardly ever 1—3 most of the time
I could not get ‘going.’ Hardly ever 1—3 most of the time
Emotional
Social
Support
On the whole, how much do your friends make
you feel loved and cared for?
A great deal 1—5 not at all
How much are your friends and relatives willing
to listen when you need to talk about your worries or problems?
A great deal 1—5 not at all

Note: This table represents the items from the Symptoms Checklist 90-Revised, the Center for Epidemiological Studies Depression Scale, and the constructed Social Support Scale.

Center for Epidemiological Studies Depression Scale

The CES-D, a self-report questionnaire meant to evaluate depressive symptoms in the general population [68], was delivered during all 3 waves. The original scale had 20-items, however, an 11-item version was used here. See Table 1 for items. The 11-item version was deemed preferable for this study because the full scale was found to be taxing for older adults [69]. Responses were measured on a three point Likert scale from 1 (hardly ever) to 3 (most of the time). The 20-item scale showed good convergent validity when compared to the SCL-90-R depression scale (r = .73-.89) [70], the BDI, and the MMPI-II [71]. The 11-item subscale also has been found to have good construct validity [72]. In the current sample, internal consistency of this scale was high (α = .84) [62].

Nevertheless, the 11-item CES-D contains questions that are not specific to depressive symptoms. For example, it also measures social relationships (i.e. people being unfriendly, people disliking them, and loneliness) and symptoms that overlap with anxiety (i.e. restlessness, appetite disturbance). To ensure that these items did not undermine the theoretical constructs, they were removed for the primary analyses (note that additional analyses were also conducted with the original 11-item CES-D; see planned analyses below). The remaining 6-item version still had high internal consistency (α = 0.80).

Perceived Emotional Social Support Scale

Two items were used to measure perceived emotional social support: (1) “On the whole, how much do your friends make you feel loved and cared for?” and (2) “How much are your friends and relatives willing to listen when you need to talk about your worries or problems?” This scale was given during Wave 2, 18-months after the loss of a spouse. Internal consistency of this scale was high (α = .80) [62]. Responses were measured on a five point Likert scale from 1 (a great deal) to 5 (not at all). Items were then reverse coded, such that higher scores indicated higher levels of social support.

Although this scale was developed for this study specifically, it shares a great deal of overlap between previously validated scales. In particular, it is similar to (1) The Medical Outcomes Study (MOS) Social Support [which likewise assesses social support using items such as “love and affection”, “listen to you”, “confide in”, and “share worries with”; 73]; (2) The Duke-UNC Functional Social Support Questionnaire [also using the following similar items “Chances to talk with someone about [all types] of problems” and “Love and affection”; 74], and (3) the Interview Schedule for Social Interaction [similarly to the current study it asks “Do you have someone you can share your most private feelings with (confide in) or not?” as well as multiple items assessing the perceived love and care; 75; 76]. Thus, although we used a novel two-item scale, it contained high item content validity with previously validated scales.

Covariates

Note that the analyses were repeated with and without controlling for the following sources of stress. During the second wave, persons were asked if they (1) experienced a life-threatening illness or injury, (2) have a serious but not life-threatening illness, (3) were robbed or burglarized, (4) involuntarily lost a job for reasons other than retirement, (5) have serious financial problems or difficulties, and (6) moved to a new residence.

Planned Analyses

All primary analyses used Bayesian structural equation models (BSEM) in Mplus 7. BSEM has unique advantages over frequentist SEM: (1) unlike frequentist SEM, which usually requires considerably larger sample sizes, BSEM coverage rates have been shown to perform well in simulation studies with sample sizes ranging from 25 to 1000 [77; 78], (2) BSEM does not have problems with non-convergence, nonsensical values, and Heywood cases [79; 80], and (3) BSEM tends to perform well under conditions of multicollinearity [81; 82]. Notably, BSEM also handles ordinal data well, allowing distributions to be directly set within the modeling framework [83]. Missing data was handled through random forest multiple imputation using the missForest package, which has greater accuracy than other multiple imputation methods [84].

Prior to the primary analyses, to ensure that there were not baseline differences between completers and non-completers, a Bayesian independent samples t-test was conducted on the factor scores of the anxiety and depression variables at Wave 1 using JASP [85]. Note that the test statistic in a Bayesian t-test is the Bayes factor. A Bayes factor is a model-based estimate of the odds of the alternative hypothesis being true compared to the null hypothesis. A Bayes Factor ranges from 0 to infinity, and the higher the Bayes factor the more evidence there is to reject the null hypothesis (i.e. that there are no differences between completers and non-completers). Values less than 1 suggest that there is more support for the null-hypothesis than there is for the alternative hypothesis [86]. Note that the default non-informative Cauchy prior was used [85].

First, we tested the first and second hypotheses that anxiety symptoms at Wave 1 would predict depressive symptoms at Wave 3, controlling for Wave 1 anxiety symptoms, and depressive symptoms at Wave 1 would predict anxiety symptoms at Wave 3, controlling for Wave 1 depression symptoms. Item residuals between Wave 1 and Wave 3 were allowed to covary. Next, we tested the hypothesis that the relationship between anxiety symptoms at Wave 1 and depressive symptoms at Wave 3 would be mediated by perceived emotional social support, controlling for Wave 1 depression symptoms (we also tested social support mediating the relationship between depression and later anxiety, controlling for earlier anxiety). Mediation was estimated directly using indirect and total effect model parameters, and these parameters were estimated directly using the Markov Chain Monte Carlo (MCMC) estimates. As with other mediation approaches (i.e. bootstrapping), BSEM parameter estimates do not make any distributional assumptions of the parameters in the estimation [77].

Model fit was assessed based on the posterior predictive p-value (PPP) [87; 88], which uses the likelihood-ratio chi-square test of the estimated data against the observed data. Based on simulation work, PPP values above 0.050 represent good model fit [83; 87]. Instead of p-values, which have been shown to be highly unreliable [89], BSEM uses Bayesian credible intervals wherein the model suggests that the true parameter has a 95% chance of falling within the specified credible interval bounds [90] and is determined to be significant if credible intervals do not contain 0. Non-informative priors were used in the modeling approach as recommended by Asparouhov and Muthen [83] and Muthén and Asparouhov [87]. All regression coefficients are also presented with a Cohen’s d effect size based on the following formula, d=βSD(β)*2n [91], and consequently the magnitude of the effect size suggests that 0.2 represents a small effect, 0.5 represents a medium effect, and over 0.8 represents a large effect [92].

All primary analyses were conducted using the CES-D items that did not overlap with emotional social relationships or with anxiety (see CES-D section above). However, to be comprehensive, all analyses were repeated with the 11-item CES-D scale. Further, to ensure that the results were not driven by experiencing other sources of stress, all analyses were repeated using each stress variable as a covariate in predicting anxiety and depression at wave 3. Note that both sets of results are reported in footnotes below.

Results

Differences between Completers and Non-Completers

For baseline anxiety, the results suggested that it was unlikely that there was a difference (BF = 0.764) between completers (M = 12.370, SD = 3.070) and non-completers (M = 13.380, SD = 4.647). Likewise, for depression the results suggested that it was unlikely that there was a difference (BF = 0.144) between completers (M = 11.04, SD = 1.707) and non-completers (M = 11.00, SD = 1.707).

Hypotheses 1-2: Anxiety Predicts Later Depression and Depression Predicts Later Anxiety

The first analysis examined our first and second hypotheses. The model demonstrated good fit (PPP = .100). Supporting our first hypothesis, anxiety symptoms significantly positively predicted subsequent depressive symptoms when controlling for baseline depression symptoms (B = 0.331, CI = 0.004 - 0.682, d= 0.271; see Figure 1).2,3 Likewise, supporting our second hypothesis, depressive symptoms significantly predicted later anxiety symptoms when controlling for baseline depression symptoms (B = 0.330, CI = 0.027 - 0.632, d = 0.301).

Figure 1.

Figure 1

In this figure depicts the first model results where anxiety and depressive symptoms at wave 1 predict anxiety and depressive symptoms at wave 3. Solid lines represent significant connections, whereas dotted lines represent insignificant connections.

Hypotheses 3-4: Emotional Social Support as a Mediator

The next set of analyses examined our third and fourth hypotheses. The model demonstrated good fit (PPP = .058). The total effect of anxiety at Wave 1 on depression at Wave 3 was significant (B = 0.368, CI = 0.009 - 0.774, d = 0.274), suggesting that anxiety continued to predict later depression when emotional social support was included in the model. Supporting our third hypothesis, anxiety symptoms significantly (B = -0.288, CI = -0.587— -0.005, d = -0.275) negatively predicted later emotional social support, and social support significantly (B = -0.496, CI = -0.711— -0.297, d= -0.662) negatively predicted later depressive symptoms (see Figure 2). Further, the indirect effect of anxiety on depression through emotional social support was significantly positive (B = 0.138, CI = 0.003 - 0.308, d = 0.253), suggesting that perceived emotional social support significantly mediated the relationship between anxiety symptoms and later depressive symptoms. Additionally, the direct relationship between anxiety symptoms and later depressive symptoms was no longer significant (B = 0.227, CI = -0.122—0.609, d = 0.176). Based on the indirect and total effect estimates, emotional social support mediated 38% of the total variance in anxiety predicting later depression. These results suggest that low emotional social support mediates a substantial portion of the variation between early anxiety and later depression.

Figure 2.

Figure 2

In this figure depicts the second model results where social support mediates at wave 2 the relationship between anxiety at wave anxiety and depressive symptoms at wave 3. Solid lines represent significant connections, whereas dotted lines represent insignificant connections.

Supporting our fourth hypothesis, in contrast to social support significantly mediating the relationship between anxiety and later depression, the relationship between earlier depression and later social support was not significant (B = -0.118, CI = -0.405 – 0.158, d = -0.143), and likewise, social support did not significantly predict later anxiety (B = -0.055, CI = -0.247 – 0.137, d = -0.080). Although the total effect of depression on later anxiety remained significant with social support in the model (B = 0.356, CI = 0.057 - 0.698, d = 0.311), the indirect effect of depression on later anxiety mediated by social support was not significant (B = 0.003, CI = - 0.003 – 0.051, d = 0.022). Thus, social support uniquely mediated the relationship between anxiety and later depression, and not vice versa.4,5

Discussion

Supporting prior research using bereaved samples [5; 16] and the wider literature of anxiety and later depressive symptoms [20; 2226; 28], the current results indicated that higher anxiety symptoms predicted higher depressive symptoms 48-months post bereavement. Additionally, depression following bereavement predicted anxiety symptoms 48-months later, suggesting a bi-directional relationship between anxiety and depressive symptoms post-bereavement. Furthermore, higher anxiety symptoms predicted lower emotional social support and lower emotional support predicted higher depressive symptoms. Thirty-eight percent of the relationship between earlier anxiety symptoms and later depressive symptoms was explained by the latter analysis. Nonetheless, perceived emotional social support failed to mediate the relationship between depression and later anxiety.

Findings in this age group may be particularly noteworthy given the unique developmental contexts of older adulthood. Older adults are more likely to experience declining physical health [93], bereavement [94], greater social isolation [95], and a loss of social support [96]. During such a pivotal time, anxious reactions to spousal loss are common [39], as the loss of one’s spouse often signifies the beginning of feeling socially isolated [32; 97]. Supporting prior theories that anxiety reactions to bereavement in older adulthood lead to social withdrawal to conserve one’s energy for personal coping [31], and contextualizing findings that less well-adjusted reactions to grief evoke frustration and low compassion from others [49], the current findings suggest that anxiety following bereavement substantially contributes to one’s level of social support during this pivotal time. Paired with findings that some older adults attempt to avoid anything that reminds them of their spouse in order to cope with spousal loss [98], future research should examine social avoidance, withdrawal, and interpersonal rejections as potential mechanisms between anxiety and low emotional social support following spousal loss in older adults.

Building on the broad literature supporting the importance of social support following bereavement and in older adulthood [10; 99], the current research also found that low emotional social support predicted symptoms of high depression two and a half years later. It is possible that the relationship between social support and later depression in bereaving older adults occurs due to those with low social support having insufficient social outlets to cope and process the loss of their spouse, and lead to feelings of loneliness, isolation [21; 33; 34], and emotional isolation [21]. Thus, future research should examine loneliness, and emotional isolation as potential pathways between low social support and later depressive symptoms in widows.

The present findings also relate to coping with stressful life events. According to theory, critical life events require major readjustment. Intensity of stress depends on whether the demands of a situation exceed individuals’ emotional coping resources [100]. Additionally, according to Holmes and Rahe [101], the death of a spouse is the most stressful life change event because it requires the most adaptive coping behaviors and is accompanied by the most psychological distress. One study found that 14-months after spousal bereavement, about a third of bereaved individuals had difficulties developing new intimate relationships, and the problem was particularly salient among older adults [102]. A similar study found that 25 months later, 37% of widowers and 58% of widows reported difficulties developing new relationships [103]. Furthermore, number of family and friends in the social network and visits from family members tend to decline over the first year and half of bereavement [57]. Our findings add to prior findings suggesting that higher anxiety may be related to such social support decline [104].

Our results support prior findings that elevated anxiety [39] and depressive symptoms [7; 914] may hold particular importance following a loss, especially in regard to the social adjustment of the widow or widower. Additionally, social support may act as a protective factor against potentially adverse effects of stressful life events, as has been found previously; specifically, high levels of social support were associated with low depressive symptoms [10; 12; 43; 44]. Therefore, social support is particularly important for protecting against psychological distress following what is considered the most stressful life event, spousal bereavement. Prior studies also found that bereavement increased the need for social support [105]. Additionally, those who had lost a spouse but perceived that they were more socially supported were less distressed than those who felt less socially supported [106108]. These results may suggest that interventions for bereaved older adults should include a component targeted toward reducing anxiety symptoms and fostering approach strategies, rather than avoidance strategies. They also suggest that targeting social support may be helpful to bereaved individuals, especially those exhibiting anxiety symptoms. In addition, because we found this relationship within the first 4 years’ post-bereavement, it is possible that anxiety may rapidly progress toward later depressive symptoms. Thus, intervention programs may be especially important during the first four years after loss of a spouse for older adults.

These results are consistent with and incrementally extend prior findings, as well as corroborate literature showing that anxiety and depressive symptoms bi-directionally predict one another years later [20; 21]. This is the first time a temporal mechanism between anxiety and depression has been examined in an older adult sample; all prior studies used adolescent or young adult samples. Nevertheless, in younger populations outside the context of bereavement, prior research found four constructs that partially longitudinally mediated the relationship between anxiety and depressive symptoms: avoidance, sociability, interpersonal oversensitivity, and perceptions of close and group relationships [26; 28; 45]. The present findings add poor interpersonal relationships to this list, suggesting that perceived emotional support may be a mechanism by which anxiety predicts later depression. Furthermore, in pairing prior literature with the current results, the depth of one’s social connections may be as influential as breadth in mediating the relationship between anxiety and later depression.

Although this study makes important contributions to the bereavement literature as well as the broader anxiety and depression literature, it is not without limitations. Additionally, the current research only utilized self-report measures, and, consequently, the results could be impacted by potential response biases. Consequently, the results should be replicated using multi-trait multimethod research with multiple informants. In particular, although we found a bi-directional longitudinal relationship between anxiety and depression following bereavement, we only found that social support unidirectionally mediated anxiety and later depression. Although little is known about the relationship between depression and later anxiety, future research should examine behavioral activation and avoidance as potential meditational candidates. In particular, depression was associated with low behavioral activation [109]. Decreases in behavioral activation could thereby decrease exposure to novel situations and experiences [109] and further exacerbate fear and avoidance cycles [109].

The current study sample also was not diverse, consisting primarily of heterosexual Caucasian females. Although a prior meta-analysis found that the relationship between anxiety and depressive symptoms was robust across gender and ethnicity [20], more research is needed to determine whether the mediating role of social support applies across different gender identities, sexual orientations, and ethnicities. We were also unable to control for other third-variables that could have connections with the constructs examined here (i.e. avoidance, neuroticism, coping techniques, religiosity), and, consequently, future work should examine perceived emotional social support while also examining other potentially related control variables. Further, the current study used an emotional social support measure that has not been used in prior work, and, consequently, future work should attempt to replicate our findings with a validated measure. Moreover, although the study established emotional social support as mediating a large portion of the variance between anxiety and later depressive symptoms following bereavement, the current study could not establish mechanisms between anxiety and social support or between social support and depression. Likewise, although mediation provides a framework to examine mechanisms between processes, it should be noted that this does not imply that the statistical findings represent causal relationships, but rather a pattern of longitudinal findings that are indicative of risk factors. Notably, although the current use of structural equation modeling allows one to account for item-level measurement error from the examination of the relationship between constructs, it sacrifices the uniqueness of the importance of some items in favor of the shared covariance. Thus, future research should examine potential mediators between anxiety symptoms and social support, such as avoidance, and social support and depressive symptoms, such as loneliness.

Table 2.

Scale Correlations and Descriptive Statistics

1 2 3 4 5
1. Anxiety Wave 1 1
2. Depression Wave 1 0.40 1
3. Emotional Social Support Wave 2 0.29 0.08 1
4. Anxiety Wave 3 0.23 0.20 0.10 1
5. Depression Wave 3 0.35 0.33 0.23 0.38 1
M 12.99 11.04 3.74 11.16 10.44
SD 4.11 1.62 1.83 2.41 1.15
Range 10–31 6–17 2–10 10–27 6–15

Note: This table first presents correlations between items in the first five rows of the table. Following this, the mean, standard deviation, and range of each of the scale measures.

Highlights.

  • Anxiety immediately following bereavement predicts depression four years later.

  • Depression immediately following bereavement predicts anxiety four years later.

  • Social support mediates the relationship between anxiety and depression

Acknowledgments

The availability of these data is made possible by a grant from the National Institute on Aging (Randolph M. Nesse, Principal Investigator, AG15948-01). The original data collection for the CLOC study was supported by NIA grants (Camille B. Wortman, Principal Investigator, AG610757-01, and James S. House, Principal Investigator, AG05561-01).

Footnotes

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2

Note that the model including the 11-item CES-D also had good fit (PPP = .058). Also, the pattern of anxiety predicting depression was consistent with the primary findings, as anxiety also significantly (B = 0.387, CI = 0.079 - 0.737, d = 0.330) positively predicted later depression in using the 11-item CES-D. Likewise, the results also showed that the model using the 11-item CES-D was consistent in that depression at wave 1 significantly predicted anxiety at wave 3 (B = 0.468, CI = 0.154 - 0.873, d = 0.361).

3

Note that the model including the stress covariates also had good fit (PPP = .058). Also, the pattern of anxiety predicting depression was consistent with the primary findings, as anxiety also significantly (B = 0.410, CI = 0.807 - 0.387, d = 0.290) positively predicted later depression while controlling for later stress. Likewise, the results also showed that the model using the stress covariates was consistent in that depression at wave 1 significantly predicted anxiety at wave 3 (B = 0.444, CI = 0.115 - 0.780, d = 0.361).

4

Note that the model including the 11-item CES-D had good fit (PPP = .058). The pattern of emotional social support mediating the relationship between anxiety and depression held, as anxiety significantly negatively predicted emotional social support (B = -0.291, CI = -0.586— -0.012, d = -0.282) and emotional social support significantly negatively predicted depression (B = -0.306, CI = -0.502— - 0.123, d = -0.446). Further the indirect effect of anxiety on depression, mediated by emotional social support, was also significantly positive (B = 0.083, CI = 0.003—0.208, d = 0.221). Additionally, the direct effect of anxiety on later depression was no longer significant (B = 0.282, CI = -0.032—0.622, d = 0.237). Also like the 6-item CES-D results, the results suggested that baseline depression did not significantly predict social support (B = -0.119, CI = -0.390 - 0.149, d = -0.123), and social support did not significantly predict later anxiety (B = -0.017, CI = -0.220 – 0.175, d = -0.024). The total effect of depression on anxiety (B = 0.554, CI = 0.256 - 0.895, d = 0.480) remained significant, but the indirect effect of depression on later anxiety mediated by social support (B = 0.000, CI = -0.038 – 0.041, d = 0.000) was not significant for the 11-item CES-D. Consequently, the theoretical implications of the model, including the reduced CES-D compared to the 11-item CES-D, are the same.

5

Note that the model including the stress covariates also had good fit (PPP = .058). Likewise, the pattern of emotional social support mediating the relationship between anxiety and depression held, as anxiety significantly negatively predicted emotional social support (B = -0.333, CI = -0.665— -0.050, d = -0.316) and emotional social support significantly negatively predicted depression (B = -0.633, CI = -0.872— - 0.442 3, d = -0.799). Further the indirect effect of anxiety on depression, mediated by emotional social support, was also significantly positive (B = 0.211, CI = 0.024—0.426, d = 0.301). Additionally, the direct effect of anxiety on later depression was no longer significant (B = 0.184, CI = -0.192—0.554, d = 0.140). Likewise when controlling for stress covariates, the results suggested that baseline depression did not significantly predict social support (B = -0.129, CI = -0.450 - 0.176, d = - 0.121), and social support did not significantly predict later anxiety (B = -0.184, CI = -0.390 – 0.024, d = -0.248). The total effect of depression on anxiety (B = 0.367, CI = 0.080 - 0.633, d = 0.358) remained significant, but the indirect effect of depression on later anxiety mediated by social support (B = 0.016, CI = -0.031 – 0.101, d = 0.067) was not significant when controlling for stress covariates. Consequently, the theoretical implications of the model, including the stress covariates, are the same.

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