Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1)-dependent paracrine mechanism.

VOLUME 289 (2014) PAGES 27199–27214

This article has been withdrawn by the authors due to reuse of the U6 loading controls in the right and left panels of Fig. 4A and the GAPDH loading controls in Figs. 7D and 9F. Additionally, the journal investigated the IGF1R immunoblot in Fig. 5C and the collagen immunoblot in Fig. 6C. The authors were not able to provide the original data for the collagen immunoblot in Fig. 6C.