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. 2017 May 10;69(6):1207–1216. doi: 10.1161/HYPERTENSIONAHA.117.09156

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© 2017 The Authors.

Hypertension is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 3. — NPNT (nephronectin) drives aldosterone production, likely through receptor ITGB1. A, NPNT overexpression increases protein-normalized aldosterone production, compared with vector control (n=4). B, NPNT silencing decreases protein-normalized aldosterone production, compared with nontargeting control (n=4). C, ITGB1 silencing decreases protein-normalized aldosterone production, compared with nontargeting control (n=4). Bars represent mean expression per group±SEM. Statistical analyses were conducted by Student t test. *P<0.05; **P<0.005; ***P<0.0005.