Table 2. ERV14 complementation and copy number.
Genotype | Unstable Chr. (×10−5) | Allelic rec. (×10−5) | Chr. loss (×10−4) | |||
---|---|---|---|---|---|---|
Median (Q1, Q3) | Fold stabil. | Median (Q1, Q3) | Fold stabil. | Median (Q1, Q3) | Fold stabil. | |
rad9∆ | 57 (40, 71) | 1 | 6.0 (2.8, 8.2) | 1 | 22 (16, 39) | 1 |
rad9∆ erv14 ERV14x2 | 40 (36, 44) | 1.4* | 4.0 (3.2, 12) | 1.5 | 15 (12, 22) | 1.5 |
rad9∆ ERV14x2a | 51 (43, 54) | 1.1 | 7.2 (6.4, 8.6) | 0.83 | 21 (19, 24) | 1.0 |
Complementation section: instability frequencies and genome fold stabilization of ERV14 complementation of rad9Δ erv14 normalized to rad9Δ erv14; ERV14 copy number section: instability frequencies and genome fold stabilization of rad9Δ with extra copies of ERV14, normalized to rad9Δ. Cells with a light gray background indicate genome fold stabilization increase; cells with a white background indicate decreased fold stabilization (<1.0 = increased instability) or no change in stabilization (=1.0). Statistically significant appears in boldface type. Kruskal–Wallis test, * P < 0.01.
Sample sizes: rad17∆ N = 6 one isolate; tel1∆ N = 6 one isolate; mad2∆ N = 3 three isolates; ChrV rad17∆ N = 3 three. isolates; ChrV mad2∆ N = 3 two isolates, rad9∆ N = 3 one isolate; rad9∆ ERV14x2 N = 3 one isolate.