Skip to main content
. Author manuscript; available in PMC: 2018 Sep 1.
Published in final edited form as: Epidemiology. 2017 Sep;28(5):719–727. doi: 10.1097/EDE.0000000000000704

Table 2.

Proportion of Pubertal Status (Breast and Pubic Hair Development) at Study Entry and End, by Smoke Exposures

Exposurea At entry to study
At end of study periodb
B2+ (15%) PH2+ (12%) B2+ (89%) PH2+ (86%)
Secondhand smoke
  None 89 (13%) 64 (9%) 631 (90%) 612 (87%)
  Low/Moderate 54 (16%) 48 (14%) 301 (88%) 290 (84%)
  High 23 (27%) 19 (22%) 72 (85%) 71 (84%)
Maternal prenatal smoking
  None 150 (14%) 115 (11%) 929 (90%) 899 (87%)
  <5cigs/day 12 (19%) 13 (20%) 51 (80%) 50 (78%)
  ≥5cigs/day 4 (13%) 3 (10%) 25 (83%) 25 (83%)
Child Cotininec (baseline)
  Q1: ≤0.25 ug/g 24 (8%) 23 (8%) 263 (92%) 255 (89%)
  Q2: 0.26–0.61 ug/g 40 (14%) 34 (12%) 252 (90%) 250 (89%)
  Q3: 0.62–2.24 ug/g 46 (16%) 28 (10%) 249 (89%) 235 (84%)
  Q4: ≥2.25 ug/g 56 (20%) 46 (16%) 241 (85%) 234 (83%)
a

For total N’s by category (denominators), see Table 1

b

Includes girls lost to follow-up before reaching puberty, as well as those not pubertal by last exam.

c

Urinary cotinine, corrected for creatinine

B2+ indicates onset of breast development, defined as Tanner stage 2 or higher; PH2+, onset of pubic hair development, defined as Tanner stage 2 or higher