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. 2017 Oct 11;8(53):90781–90795. doi: 10.18632/oncotarget.21796

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Copyright: © 2017 Li et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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A., B. Protein expression A. of PARP, active caspase-3 and Bcl-2; and their gene expression B. in chondrocytes, as estimated by Western blot and qPCR. Cultures were treated with 10 ng/ml IL-1β for 0, 4, 8, 12 and 24 h or co-treated with 50 μM baicalein and 10 ng/ml IL-1β for 0, 12, 24, 36 and 48 h after exposure to 10 ng/ml IL-1β alone for 24 h. Values are shown as the means ± SEM (n = 5). * P < 0.05;** P < 0.01;*** P < 0.001.

A., B. Protein expression A. of PARP, active caspase-3 and Bcl-2; and their gene expression B. in chondrocytes, as estimated by Western blot and qPCR. Cultures were treated with 10 ng/ml IL-1β for 0, 4, 8, 12 and 24 h or co-treated with 50 μM baicalein and 10 ng/ml IL-1β for 0, 12, 24, 36 and 48 h after exposure to 10 ng/ml IL-1β alone for 24 h. Values are shown as the means ± SEM (n = 5). * P < 0.05;** P < 0.01;*** P < 0.001.