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. 2017 Oct 26;45(21):12600. doi: 10.1093/nar/gkx1099

Autoregulatory circuit of human rpL3 expression requires hnRNP H1, NPM and KHSRP

Annapina Russo 1,2, Morena Catillo 1, Davide Esposito 1, Paola Briata 3, Concetta Pietropaolo 1, Giulia Russo 1,*
PMCID: PMC5716608  PMID: 29087476

Nucleic Acids Research (2011) 39(17) 7576–7585, https://doi.org/10.1093/nar/gkr461

The authors wish to make the following correction to their article:

A β-actin control band, belonging to panel B, has accidentally been duplicated in panel A. A new Figure 5 is provided below, the correct β-actin control bands in panel A are marked in red.

Figure 5.

Figure 5.

(A) Effects of RNAi-mediated depletion of hnRNP H1 on rpL3-a mRNA levels in L3–8 cells. Representative RT-PCR analysis of total RNA from CHX-treated L3–8 cells, untransfected or transfected with hnRNP H1-siRNA, NI or induced I for HA-rpL3 expression. (B) Effects of RNAi-mediated depletion of hnRNP H1 and KHSRP overexpression on rpL3-a mRNA levels in L3–8 cells. Representative RT–PCR analysis of total RNA from CHX-treated L3–8 cells untransfected or cotransfected with hnRNP H1-siRNA and Flag-KHSRP, NI or induced I for HA-rpL3 expression. The levels of rpL3-a mRNA were quantified by PhosphorImager (Bio-Rad) and normalized to β-actin levels.


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